Liver failure in protoporphyria: long-term treatment with oral charcoal. 1999

A Gorchein, and G R Foster

UI MeSH Term Description Entries
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D002606 Charcoal An amorphous form of carbon prepared from the incomplete combustion of animal or vegetable matter, e.g., wood. The activated form of charcoal is used in the treatment of poisoning. (Grant & Hackh's Chemical Dictionary, 5th ed) Activated Charcoal,Actidose,Actidose-Aqua,Adsorba,Carbomix,Charbon,CharcoAid,CharcoCaps,Charcodote,Formocarbine,Insta-Char,Kohle-Compretten,Kohle-Hevert,Kohle-Pulvis,Kohle-Tabletten Boxo-Pharm,Liqui-Char,Norit,Ultracarbon,Charcoal, Activated
D004334 Drug Administration Schedule Time schedule for administration of a drug in order to achieve optimum effectiveness and convenience. Administration Schedule, Drug,Administration Schedules, Drug,Drug Administration Schedules,Schedule, Drug Administration,Schedules, Drug Administration
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000284 Administration, Oral The giving of drugs, chemicals, or other substances by mouth. Drug Administration, Oral,Administration, Oral Drug,Oral Administration,Oral Drug Administration,Administrations, Oral,Administrations, Oral Drug,Drug Administrations, Oral,Oral Administrations,Oral Drug Administrations
D017093 Liver Failure Severe inability of the LIVER to perform its normal metabolic functions, as evidenced by severe JAUNDICE and abnormal serum levels of AMMONIA; BILIRUBIN; ALKALINE PHOSPHATASE; ASPARTATE AMINOTRANSFERASE; LACTATE DEHYDROGENASES; and albumin/globulin ratio. (Blakiston's Gould Medical Dictionary, 4th ed) Hepatic Failure
D017121 Porphyria, Hepatoerythropoietic An autosomal recessive cutaneous porphyria that is due to a deficiency of UROPORPHYRINOGEN DECARBOXYLASE in both the LIVER and the BONE MARROW. Similar to PORPHYRIA CUTANEA TARDA, this disorder is caused by defects in the fifth enzyme in the 8-enzyme biosynthetic pathway of HEME, but is a homozygous enzyme deficiency with less than 10% of the normal enzyme activity. Cutaneous lesions are severe and mutilating. Hepatoerythropoietic Porphyria,Porphyria, Erythrohepatic,Erythrohepatic Porphyria,Erythrohepatic Porphyrias,Hepatoerythropoietic Porphyrias,Porphyrias, Erythrohepatic,Porphyrias, Hepatoerythropoietic

Related Publications

A Gorchein, and G R Foster
December 2002, Photodermatology, photoimmunology & photomedicine,
A Gorchein, and G R Foster
June 1999, Journal of gastroenterology,
A Gorchein, and G R Foster
March 1996, Journal of the American Academy of Dermatology,
A Gorchein, and G R Foster
January 2005, Dermatology (Basel, Switzerland),
A Gorchein, and G R Foster
September 1982, Archives of dermatology,
A Gorchein, and G R Foster
April 1999, European journal of gastroenterology & hepatology,
A Gorchein, and G R Foster
June 1994, Lancet (London, England),
A Gorchein, and G R Foster
July 1994, Lancet (London, England),
A Gorchein, and G R Foster
July 1994, Lancet (London, England),
A Gorchein, and G R Foster
October 1994, Lancet (London, England),
Copied contents to your clipboard!