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Crossing over was measured on the normally achiasmate fourth chromosome in females homozygous for one of our different recombination-defective meiotic mutants. Under the influence of those meiotic mutants that affect the major chromosomes by altering the spatial distribution of exchanges, meiotic fourth-chromosome recombinants were recovered irrespective of whether or not the meiotic mutant decreases crossing over on the other chromosomes. No crossing over, on the other hand, was detected on chromosome 4 in either wild type or in the presence of a meiotic mutant that decreases the frequency, but does not affect the spatial distribution, of exchange on the major chromosomes. It is concluded from these observations that (a) in wild type there are regional constraints on exchange that can be attenuated or eliminated by the defects caused by recombination-defective meiotic mutants; [b] these very constraints account for the absence of recombination on chromosome 4 in wild type; and [c] despite being normally achiasmate, chromosome 4 responds to recombination-defective meiotic mutants in the same way as do the other chromosomes.
L Sandler, and
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March 1974,
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L Sandler, and
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July 1979,
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L Sandler, and
P Szauter
October 1982,
Proceedings of the National Academy of Sciences of the United States of America,
L Sandler, and
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January 1973,
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L Sandler, and
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January 1984,
Genetics,
L Sandler, and
P Szauter
July 1951,
Proceedings of the National Academy of Sciences of the United States of America,
L Sandler, and
P Szauter
June 2008,
Genetics research,
L Sandler, and
P Szauter
February 2012,
G3 (Bethesda, Md.),
L Sandler, and
P Szauter
June 2001,
Genetika,
L Sandler, and
P Szauter
September 1936,
Genetics,
