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Clinical, electrocardiographic and laboratory data were found out in 52 patients with cardiac arrhythmias and conduction defects due to digoxin intoxication. Forty six nontoxic patients were also studied for comparison. Blood urea concentration was significantly higher in toxic patients as compared to nontoxic ones (P less than 0-01). Ventricular bigeminy and trigeminy (38.6%), multifocal ventricular premature beats (25%) and second or third degree A-V blocks (25%) were very much prevalent. Fairly good correlations have been observed between different cardiac arrhythmias and serum digoxin levels. Significantly higher mean serum digoxin levels were observed in patients with A-V block and multifocal ventricular premature beats as compared to patients with supraventricular arrhythmias.
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
February 1982,
Drug intelligence & clinical pharmacy,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
February 1974,
The Journal of pediatrics,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
December 1970,
The Journal of clinical investigation,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
November 1981,
Arizona medicine,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
October 1973,
Acta medica Scandinavica,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
December 1987,
Clinical pharmacology and therapeutics,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
January 1984,
Zeitschrift fur Rechtsmedizin. Journal of legal medicine,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
May 1971,
The New England journal of medicine,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
January 1975,
Acta cardiologica,
R B Singh, and
G P Dube, and
K P Dube, and
B C Katiyar
March 1977,
Deutsche medizinische Wochenschrift (1946),
