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Placental blood-flow rates in rabbits were measured by the indirect method of local apparent conductivity. In our experiments, hypertension was produced by injection or infusion of hypertensive drugs. As a result of hypertension we observed a marked decrease in placental blood flow. Vasoconstriction seems to be the pathophysiologic mechanism causing the reduced blood flow rate. Accordingly, the placenta may not be considered as a "priviledged organ" in circulatory regulations. In our experiments, placental ischemia subsequently to ligatures did not produce hypertension so that the production of a placental pressor-substance could not be demonstrated. If we assume a circulus vitiosus hypertension-placental ischemia-hypertension, the hypertension seems to be the primary cause.
H Kraus, and
R H Borst, and
R Schuhmann
November 1973,
Der Anaesthesist,
H Kraus, and
R H Borst, and
R Schuhmann
January 1981,
Zentralblatt fur Chirurgie,
H Kraus, and
R H Borst, and
R Schuhmann
January 1977,
Arzneimittel-Forschung,
H Kraus, and
R H Borst, and
R Schuhmann
January 1978,
Journal de gynecologie, obstetrique et biologie de la reproduction,
H Kraus, and
R H Borst, and
R Schuhmann
February 1974,
Deutsche medizinische Wochenschrift (1946),
H Kraus, and
R H Borst, and
R Schuhmann
May 1981,
Nihon Shokakibyo Gakkai zasshi = The Japanese journal of gastro-enterology,
H Kraus, and
R H Borst, and
R Schuhmann
August 1978,
Radiologia diagnostica,
H Kraus, and
R H Borst, and
R Schuhmann
October 1976,
Archiv fur orthopadische und Unfall-Chirurgie,
H Kraus, and
R H Borst, and
R Schuhmann
April 1981,
Immunitat und Infektion,
H Kraus, and
R H Borst, and
R Schuhmann
December 1979,
Die Pharmazie,
