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Insulin resistance (IR), probably a common pathway of atherosclerosis development in various diseases, was suggested to be related to magnesium (Mg) deficiency. The in vivo observations required an in vitro extension. The study was performed on isolated rat soleus muscle incubated in Ringer bicarbonate with/without Mg. Mg deficiency inhibited basal, insulin- and tolbutamide-stimulated glucose utilization. Insulin-stimulated glucose utilization was inhibited even in the case that insulin was given to rats before sacrifice. Similar inhibition of glucose utilization was found in Ca deficiency and the simultaneous lack of Mg had no additive effect. It is concluded that Mg deficiency inhibits glucose utilization at the level of Ca mediation of glucose transport regulation.
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
October 1990,
FEBS letters,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
July 1988,
The American journal of physiology,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
September 1987,
Diabetes,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
September 1987,
Pflugers Archiv : European journal of physiology,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
October 2006,
Proceedings of the National Academy of Sciences of the United States of America,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
August 1995,
The international journal of biochemistry & cell biology,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
October 2010,
Aging,
K Stefíková, and
V Spustová, and
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R Dzúrik
July 2004,
Journal of applied physiology (Bethesda, Md. : 1985),
K Stefíková, and
V Spustová, and
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R Dzúrik
February 1998,
Molecular genetics and metabolism,
K Stefíková, and
V Spustová, and
K Sebeková, and
R Dzúrik
June 2009,
Biological trace element research,
