-transparent.png){kind=logo}
The purpose of this study was to investigate different forms of long-term potentiation (LTP) induced by different compounds. As shown in results, the influx of calcium via voltage-dependent calcium channels (VDCCs) was responsible for ( - )-clausenamide-induced LTP in hippocampus, while Rg1 induced LTP is mediated by the entry of calcium via N-methyl-d-aspartate glutamate (NMDA) receptor. The findings in the present study provide pharmacological basis for the nootropic mechanisms of these two compounds.
Related Publications
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
January 1998,
Neurobiology (Budapest, Hungary),
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
June 2008,
The Journal of physiology,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
October 1990,
Nature,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
November 1996,
Journal of neurophysiology,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
January 1997,
Reviews in the neurosciences,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
September 1995,
Nature,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
June 1990,
Science (New York, N.Y.),
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
April 2008,
The Journal of physiology,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
February 2013,
The Journal of physiology,
L Xu, and
X-Y Wang, and
S-L Liu, and
J-T Zhang
February 1991,
Science (New York, N.Y.),
