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Typical herpetic papulovesicular skin lesions developed in an apparently normal infant at 12 days of age and were followed within 48 hours by signs and symptoms of acute encephalitis. Herpes simplex virus type 2 was cultured from the intact skin vesicles, and a fourfold increase in complement fixation titer to herpes simplex virus type 2 was found over the ensuing 24 days. The infant survived her acute illness, but was left with severe neurologic sequelae manifested as microcephaly with multicystic cerebral degeneration. The short-term and convalescent course is documented by serial, clinical, and EEG examinations, and the nature of the cerebral damage is demonstrated by computerized transaxial tomography.
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
March 1988,
Lancet (London, England),
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
June 1984,
The Journal of the Kentucky Medical Association,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
April 1995,
Indian pediatrics,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
July 1984,
No to hattatsu = Brain and development,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
January 1990,
Acta neuropathologica,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
August 1980,
Tidsskrift for den Norske laegeforening : tidsskrift for praktisk medicin, ny raekke,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
June 1993,
Journal of the Royal Army Medical Corps,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
February 2003,
Nihon rinsho. Japanese journal of clinical medicine,
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
August 2002,
Medizinische Klinik (Munich, Germany : 1983),
J B Smith, and
R V Groover, and
D W Klass, and
O W Houser
January 1982,
JAMA,
