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Pathological aspects of lipid peroxidation. 2010
Anne Negre-Salvayre, and
Nathalie Auge, and
Victoria Ayala, and
Huveyda Basaga, and
Jordi Boada, and
Rainer Brenke, and
Sarah Chapple, and
Guy Cohen, and
Janos Feher, and
Tilman Grune, and
Gabriella Lengyel, and
Giovanni E Mann, and
Reinald Pamplona, and
Giuseppe Poli, and
Manuel Portero-Otin, and
Yael Riahi, and
Robert Salvayre, and
Shlomo Sasson, and
Jose Serrano, and
Ofer Shamni, and
Werner Siems, and
Richard C M Siow, and
Ingrid Wiswedel, and
Kamelija Zarkovic, and
Neven Zarkovic
INSERM I2MR-858, IFR-150, Avenue J Poulhès, BP 84225, Toulouse, France. anne.negre-salvayre@inserm.fr
Lipid peroxidation (LPO) product accumulation in human tissues is a major cause of tissular and cellular dysfunction that plays a major role in ageing and most age-related and oxidative stress-related diseases. The current evidence for the implication of LPO in pathological processes is discussed in this review. New data and literature review are provided evaluating the role of LPO in the pathophysiology of ageing and classically oxidative stress-linked diseases, such as neurodegenerative diseases, diabetes and atherosclerosis (the main cause of cardiovascular complications). Striking evidences implicating LPO in foetal vascular dysfunction occurring in pre-eclampsia, in renal and liver diseases, as well as their role as cause and consequence to cancer development are addressed.
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Janos Feher, and
Tilman Grune, and
Gabriella Lengyel, and
Giovanni E Mann, and
Reinald Pamplona, and
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Jose Serrano, and
Ofer Shamni, and
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Sarah Chapple, and
Guy Cohen, and
Janos Feher, and
Tilman Grune, and
Gabriella Lengyel, and
Giovanni E Mann, and
Reinald Pamplona, and
Giuseppe Poli, and
Manuel Portero-Otin, and
Yael Riahi, and
Robert Salvayre, and
Shlomo Sasson, and
Jose Serrano, and
Ofer Shamni, and
Werner Siems, and
Richard C M Siow, and
Ingrid Wiswedel, and
Kamelija Zarkovic, and
Neven Zarkovic
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