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The effect of tetragastrin on pancreatic tumors induced by azaserine was investigated in Wistar rats. Rats were given 25 weekly injections of 10 mg/kg body weight of azaserine and 1 mg/kg body weight of tetragastrin as a suspension in olive oil every other day. Carcinogen-induced pancreatic lesions were examined by histochemical techniques, and were classified as ATPase-positive or ATPase-negative. In week 62, quantitative histological analysis showed that prolonged administration of tetragastrin had little or no influence on the number and size of the carcinogen-induced pancreatic lesions, although it caused significantly increased cell proliferation, indicated by a greater labelling index of the pancreatic acinar cells.
M Tatsuta, and
H Iishi, and
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A Nakaizumi
June 1987,
Cancer research,
M Tatsuta, and
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March 1981,
Cancer research,
M Tatsuta, and
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February 1990,
Carcinogenesis,
M Tatsuta, and
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June 1996,
The Journal of surgical research,
M Tatsuta, and
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January 1987,
Pancreas,
M Tatsuta, and
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A Nakaizumi
January 1985,
Drug-nutrient interactions,
M Tatsuta, and
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M Baba, and
A Nakaizumi
October 1981,
Cancer research,
M Tatsuta, and
H Iishi, and
M Baba, and
A Nakaizumi
March 1998,
Pancreas,
M Tatsuta, and
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June 1992,
Cancer research,
M Tatsuta, and
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May 1993,
Pancreas,
