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Quail embryo fibroblasts were infected at low multiplicity with avian sarcoma virus, and transformed cells were selected by their ability to form colonies in agar. Five clones that failed to produce focus-forming virus were examined for (i) intactness of the integrated proviral DNA, (ii) intracellular viral RNA production, (iii) intracellular viral antigen production, (iv) production of virus particles, and (v) rescue of a functional src gene and of parental host range determinants by superinfection with Rous-associated virus-60, an avian leukosis virus of subgroup E. Deletions in the integrated viral DNA were apparent in three of the five nonproducer clones. In one clone producing focus-forming virus, analysis of the integrated viral DNA revealed an insertion in the region of the genome that codes for src.
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T W Hsu, and
C Yeater, and
J L Sabran, and
G E Mark, and
A Kaji, and
J M Taylor
November 1981,
Virology,
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J M Taylor
January 1988,
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J M Taylor
June 1982,
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C Yeater, and
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Proceedings of the National Academy of Sciences of the United States of America,
W S Mason, and
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February 1982,
Virology,
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C Yeater, and
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G E Mark, and
A Kaji, and
J M Taylor
January 1972,
Neoplasma,
W S Mason, and
T W Hsu, and
C Yeater, and
J L Sabran, and
G E Mark, and
A Kaji, and
J M Taylor
September 1970,
Virology,
