Inflammation, coronary flow reserve, and microvascular dysfunction: moving beyond cardiac syndrome X. 2013

Viviany R Taqueti, and Paul M Ridker

UI MeSH Term Description Entries
D007249 Inflammation A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function. Innate Inflammatory Response,Inflammations,Inflammatory Response, Innate,Innate Inflammatory Responses
D008297 Male Males
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D017566 Microvascular Angina ANGINA PECTORIS or angina-like chest pain with a normal coronary arteriogram and positive EXERCISE TEST. The cause of the syndrome is unknown. While its recognition is of clinical importance, its prognosis is excellent. (Braunwald, Heart Disease, 4th ed, p1346; Jablonski Dictionary of Syndromes & Eponymic Diseases, 2d ed). It is different from METABOLIC SYNDROME X, a syndrome characterized by INSULIN RESISTANCE and HYPERINSULINEMIA, that has increased risk for cardiovascular disease. Angina Pectoris with Normal Coronary Arteriogram,Angina, Microvascular,Cardiac Syndrome X,Syndrome X, Angina,Syndrome X, Cardiac,X Syndrome, Angina,Angina Syndrome X,Angina Syndrome Xs,Angina X Syndrome,Angina X Syndromes,Syndrome Xs, Angina,Syndrome, Angina X
D055806 Microvessels The finer blood vessels of the vasculature that are generally less than 100 microns in internal diameter. Microvasculature,Microvascular Network,Microvascular Networks,Microvessel,Network, Microvascular,Networks, Microvascular

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