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Syrian male hamsters weighing 80-100g were placed on either a magnesium deficient diet (MgD) or an identical diet supplemented with 100 mmols/Kg MgCl. Animals from each group received vitamin E 10, 15, and 25mg three-week slow release pellets, as subcutaneous implants. The animals were sacrificed after 14 days and their hearts isolated for morphological analysis. H&E stained sections were examined by a computer image analysis system for a morphometric determination of the severity of myocardial injury. Vitamin E significantly (p less than 0.01) reduced both the numerical density and the area fraction of MgD lesions. These data indicate possible free radical participation in the mechanism of injury.
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
December 1988,
Molecular and cellular biochemistry,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
October 1979,
The New England journal of medicine,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
January 1988,
The American journal of cardiovascular pathology,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
June 1992,
Journal of the American College of Nutrition,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
June 1994,
Free radical biology & medicine,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
April 2009,
JPMA. The Journal of the Pakistan Medical Association,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
January 1985,
Heart and vessels. Supplement,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
December 1984,
Journal of the American Veterinary Medical Association,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
July 1973,
Acta neuropathologica,
A M Freedman, and
A H Atrakchi, and
M M Cassidy, and
W B Weglicki
February 1999,
Archives of physiology and biochemistry,
