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The Venezuelan equine encephalomyelitis (VEE) is one the most serious viral infections of the nervous system. It has a wide geographic distribution and may give rise to sequela like mental retardation, amnesia, abortion, epilepsy and hidroanencephaly in infected humans and animals. The pathology of this infection is focused mainly in two tissues: lymphohematopoietic and nervous. The VEE virus has a special cytopathic activity on the nervous cells (glia and neurons) while the lesions produced in the myelin are probably a consequence of the immunological response of the host to the infection. The alterations produced by the VEE virus in different neuronal types can originate changes in the brain concentrations of several neurotransmitters and their receptors. Some biochemical modifications that have also been reported could be due to the cytopathic effect of the virus.
M E de Bellard, and
S Levine, and
E Bonilla
March 1989,
Veterinary heritage : bulletin of the American Veterinary History Society,
M E de Bellard, and
S Levine, and
E Bonilla
January 1971,
Bulletin - Office international des epizooties,
M E de Bellard, and
S Levine, and
E Bonilla
December 1977,
Teratology,
M E de Bellard, and
S Levine, and
E Bonilla
January 1974,
Advances in veterinary science and comparative medicine,
M E de Bellard, and
S Levine, and
E Bonilla
January 2004,
Annual review of entomology,
M E de Bellard, and
S Levine, and
E Bonilla
January 1967,
Advances in veterinary science,
M E de Bellard, and
S Levine, and
E Bonilla
January 1997,
Revista panamericana de salud publica = Pan American journal of public health,
M E de Bellard, and
S Levine, and
E Bonilla
November 2013,
Veterinary microbiology,
M E de Bellard, and
S Levine, and
E Bonilla
October 1995,
MMWR. Morbidity and mortality weekly report,
M E de Bellard, and
S Levine, and
E Bonilla
November 2009,
Vaccine,
