The effect of premature ventricular contraction on left ventricular relaxation, chamber stiffness, and filling in humans. 1989

M F Stoddard, and A C Pearson, and M J Kern, and A J Labovitz
Department of Internal Medicine, St. Louis University School of Medicine.

To investigate the influence of single spontaneous premature ventricular ectopic beats on left ventricular contraction, relaxation, chamber stiffness, and filling, we examined 21 patients with simultaneous micromanometer left ventricular pressure tracings and echocardiograms. Instantaneous left ventricular diameter and mitral valve inflow velocity were obtained by using of M-mode and pulsed Doppler echocardiography, respectively. The isovolumic relaxation time constants (TL and TD) were calculated by mean of a zero (TL) and variable (TD) asymptote pressure. The chamber stiffness constants were derived from the diastolic pressure-diameter (kd) and pressure-volume (kv) relationships. The extrasystolic beat was associated with marked impairments of relaxation, systolic function, and diastolic filling as seen by an increased TL (53 to 71 msec; p less than 0.001), TD (59 to 89 msec; p less than 0.005), time from maximum negative dp/dt to the lowest diastolic pressure (147 to 170 msec; p less than 0.05), and decreased number of elasped TDs (3.1 to 2.4; p less than 0.05), end-systolic pressure-diameter ratio (2.4 to 1.7; p less than 0.001), maximum positive dp/dt (1904 to 1326 mm Hg/sec; p less than 0.001), shortening fraction (31% to 21%; p less than 0.001), and peak early filling velocity (59 to 49 cm/sec; p less than 0.001). Chamber stiffness constants were unchanged. Relaxation and chamber stiffness were unchanged during the postextrasystolic beat as reflected by TL, TD, maximum negative dp/dt, and kd and kv.(ABSTRACT TRUNCATED AT 250 WORDS)

UI MeSH Term Description Entries
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D009200 Myocardial Contraction Contractile activity of the MYOCARDIUM. Heart Contractility,Inotropism, Cardiac,Cardiac Inotropism,Cardiac Inotropisms,Contractilities, Heart,Contractility, Heart,Contraction, Myocardial,Contractions, Myocardial,Heart Contractilities,Inotropisms, Cardiac,Myocardial Contractions
D003327 Coronary Disease An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels. Coronary Heart Disease,Coronary Diseases,Coronary Heart Diseases,Disease, Coronary,Disease, Coronary Heart,Diseases, Coronary,Diseases, Coronary Heart,Heart Disease, Coronary,Heart Diseases, Coronary
D003971 Diastole Post-systolic relaxation of the HEART, especially the HEART VENTRICLES. Diastoles
D004452 Echocardiography Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic. Echocardiography, Contrast,Echocardiography, Cross-Sectional,Echocardiography, M-Mode,Echocardiography, Transthoracic,Echocardiography, Two-Dimensional,Transthoracic Echocardiography,2-D Echocardiography,2D Echocardiography,Contrast Echocardiography,Cross-Sectional Echocardiography,Echocardiography, 2-D,Echocardiography, 2D,M-Mode Echocardiography,Two-Dimensional Echocardiography,2 D Echocardiography,Cross Sectional Echocardiography,Echocardiography, 2 D,Echocardiography, Cross Sectional,Echocardiography, M Mode,Echocardiography, Two Dimensional,M Mode Echocardiography,Two Dimensional Echocardiography
D004548 Elasticity Resistance and recovery from distortion of shape.
D005260 Female Females
D006321 Heart The hollow, muscular organ that maintains the circulation of the blood. Hearts
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man

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