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Somatosensory evoked potentials (SEPs) have been studied in many disease states since they were first described by Dawson in 1947. However, there have been very few reports of SEP findings in patients with spinal cord hemisection. On the basis of clinical correlation, Giblin first postulated that the activity giving rise to the initial cortical components of the SEP travels through the dorsal column-lemniscal system. However, the pathway through which these evoked potentials are transmitted has recently been questioned. This paper examines the results of tibial nerve SEPS performed on four patients with spinal cord hemisection (Brown-Séquard syndrome). In all four cases presented, the impairment of cortical SEP components was consistently associated with stimulation of the leg ipsilateral to the side of cord injury. We conclude that the most likely mechanism of transmission is through the ipsilateral dorsal columnlemniscal system.
K K Bloom, and
G Goldberg
January 1991,
Pediatric neurology,
K K Bloom, and
G Goldberg
April 1989,
American journal of veterinary research,
K K Bloom, and
G Goldberg
January 1990,
Electroencephalography and clinical neurophysiology. Supplement,
K K Bloom, and
G Goldberg
January 1982,
Advances in neurology,
K K Bloom, and
G Goldberg
January 1989,
Neurologie et psychiatrie,
K K Bloom, and
G Goldberg
October 1984,
Surgical neurology,
K K Bloom, and
G Goldberg
December 1993,
Electroencephalography and clinical neurophysiology,
K K Bloom, and
G Goldberg
January 1991,
Neurologia i neurochirurgia polska,
K K Bloom, and
G Goldberg
January 1982,
Archiv fur Psychiatrie und Nervenkrankheiten,
K K Bloom, and
G Goldberg
October 2008,
Neurophysiologie clinique = Clinical neurophysiology,
