[Conjunctival calcification in patients in chronic hemodialysis. Morphologic, clinical and epidemiologic study]. 1988

M Vignanelli, and C A Stucchi
Hôpital Ophtalmique Universitaire, Lausanne.

The authors compared the presence of conjunctival calcifications in a group of 37 patients undergoing chronic hemodialysis with laboratory findings and radiologic examinations. These calcifications appear to develop in patients suffering from hypercalcemia, particularly when the mathematical product of inorganic calcium and phosphorus (Ca x PO4) exceeds the value of 3.8-4.0. Ocular calcifications are usually asymptomatic although conjunctival inflammation due to crystal deposition, better known as "red eyes", has been reported in uremic patients. This has been noted to improve after the Ca x PO4 product is reduced. Such a conjunctival inflammation has not been reported in patients undergoing chronic hemodialysis and we do not report any case of disturbance due to the presence of conjunctival lesions. These lesions usually occur in the paralimbal conjunctiva, in the limbal area exposed by the interpalpebral fissure. This is thought to be due to the relatively high alkalinity, resulting from the diffusion of CO2 from the exposed surface of the eye, which promotes the deposition of calcium salts. However, the frequent presence of pinguecula, or histologic elastosis, in the eyes of uremic patients with calcifications, suggests a pathogenic role of local degenerative changes as well. Among these 37 patients, 6 underwent a biopsy of the conjunctival lesions, allowing a pathological examination. This permitted the authors to confirm the presence of calcium deposits in the basal lamina of the conjunctival epithelium and also in the sub-epithelial tissue. In every case, a significant degeneration of the elastic tissue of the conjunctiva was found, confirming that this lesion can precede calcium deposition in these particular patients.(ABSTRACT TRUNCATED AT 250 WORDS)

UI MeSH Term Description Entries
D007676 Kidney Failure, Chronic The end-stage of CHRONIC RENAL INSUFFICIENCY. It is characterized by the severe irreversible kidney damage (as measured by the level of PROTEINURIA) and the reduction in GLOMERULAR FILTRATION RATE to less than 15 ml per min (Kidney Foundation: Kidney Disease Outcome Quality Initiative, 2002). These patients generally require HEMODIALYSIS or KIDNEY TRANSPLANTATION. ESRD,End-Stage Renal Disease,Renal Disease, End-Stage,Renal Failure, Chronic,Renal Failure, End-Stage,Chronic Kidney Failure,End-Stage Kidney Disease,Chronic Renal Failure,Disease, End-Stage Kidney,Disease, End-Stage Renal,End Stage Kidney Disease,End Stage Renal Disease,End-Stage Renal Failure,Kidney Disease, End-Stage,Renal Disease, End Stage,Renal Failure, End Stage
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D002114 Calcinosis Pathologic deposition of calcium salts in tissues. Calcification, Pathologic,Calcinosis, Tumoral,Microcalcification,Microcalcinosis,Pathologic Calcification,Calcinoses,Calcinoses, Tumoral,Microcalcifications,Microcalcinoses,Tumoral Calcinoses,Tumoral Calcinosis
D003229 Conjunctival Diseases Diseases involving the CONJUNCTIVA. Conjunctival Disease,Disease, Conjunctival,Diseases, Conjunctival
D005260 Female Females
D006435 Renal Dialysis Therapy for the insufficient cleansing of the BLOOD by the kidneys based on dialysis and including hemodialysis, PERITONEAL DIALYSIS, and HEMODIAFILTRATION. Dialysis, Extracorporeal,Dialysis, Renal,Extracorporeal Dialysis,Hemodialysis,Dialyses, Extracorporeal,Dialyses, Renal,Extracorporeal Dialyses,Hemodialyses,Renal Dialyses
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000328 Adult A person having attained full growth or maturity. Adults are of 19 through 44 years of age. For a person between 19 and 24 years of age, YOUNG ADULT is available. Adults
D000368 Aged A person 65 years of age or older. For a person older than 79 years, AGED, 80 AND OVER is available. Elderly

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