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A potential serious complication in infants with congenital obstructive uropathy: Secondary pseudohypoaldosteronism. 2019

Sabriye Korkut, and Leyla Akin, and Nihal Hatipoglu, and Ahmet Özdemir, and Zübeyde Gündüz, and Ismail Dursun, and Levent Korkmaz, and Ismail Dursun, and Selim Kurtoglu
Department of Neonatology, Erciyes University Medical Faculty, Turkey.

Patients who have secondary pseudohypoaldosteronism (PHA) in addition to hyponatraemia, hyperpotassaemia and high serum aldosterone levels for the age were included in this retrospective study.Among eight patients, seven patients were diagnosed with PHA secondary to obstructive uropathy (OUP), whereas one patient had PHA secondary to ileostomy. Six patients with OUP had simultaneous urinary tract infection (UTI) and in all except one patient, secondary PHA recovered with only UTI treatment before applying surgical correction. All the patients were younger than 3 months age. In three patients with PUV diagnosis, salt wasting recurred in an UTI episode under 3 months of age.

UI MeSH Term Description Entries
D007010 Hyponatremia Deficiency of sodium in the blood; salt depletion. (Dorland, 27th ed) Hyponatremias
D007223 Infant A child between 1 and 23 months of age. Infants
D008297 Male Males
D009318 Natriuresis Sodium excretion by URINATION. Natriureses
D011546 Pseudohypoaldosteronism A heterogeneous group of disorders characterized by renal electrolyte transport dysfunctions. Congenital forms are rare autosomal disorders characterized by neonatal hypertension, HYPERKALEMIA, increased RENIN activity and ALDOSTERONE concentration. The Type I features HYPERKALEMIA with sodium wasting; Type II, HYPERKALEMIA without sodium wasting. Pseudohypoaldosteronism can be the result of a defective renal electrolyte transport protein or acquired after KIDNEY TRANSPLANTATION. Gordon Hyperkalemia-Hypertension Syndrome,Hyperpotassemia and Hypertension, Familial,Hypertensive Hyperkalemia, Familial,Pseudohypoaldosteronism Type 1,Pseudohypoaldosteronism Type 1, Autosomal Recessive,Pseudohypoaldosteronism, Type I,Pseudohypoaldosteronism, Type I, Autosomal Dominant,Pseudohypoaldosteronism, Type I, Autosomal Recessive,Pseudohypoaldosteronism, Type II,Familial Hyperpotassemia and Hypertension,Familial Hypertensive Hyperkalemia,Pseudohypoaldosteronism Type 1, Autosomal Dominant,Pseudohypoaldosteronism Type 2,Pseudohypoaldosteronism Type I,Pseudohypoaldosteronism Type II,Familial Hypertensive Hyperkalemias,Gordon Hyperkalemia Hypertension Syndrome,Hyperkalemia, Familial Hypertensive,Hyperkalemia-Hypertension Syndrome, Gordon,Hyperkalemias, Familial Hypertensive,Hypertensive Hyperkalemias, Familial,Pseudohypoaldosteronism Type 1s,Pseudohypoaldosteronism Type 2s,Pseudohypoaldosteronism Type IIs,Pseudohypoaldosteronism Type Is,Pseudohypoaldosteronisms,Pseudohypoaldosteronisms, Type I,Pseudohypoaldosteronisms, Type II,Syndrome, Gordon Hyperkalemia-Hypertension,Type 1, Pseudohypoaldosteronism,Type I Pseudohypoaldosteronism,Type I, Pseudohypoaldosteronism,Type II Pseudohypoaldosteronism,Type II Pseudohypoaldosteronisms,Type II, Pseudohypoaldosteronism,Type IIs, Pseudohypoaldosteronism,Type Is, Pseudohypoaldosteronism
D003937 Diagnosis, Differential Determination of which one of two or more diseases or conditions a patient is suffering from by systematically comparing and contrasting results of diagnostic measures. Diagnoses, Differential,Differential Diagnoses,Differential Diagnosis
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D006947 Hyperkalemia Abnormally high potassium concentration in the blood, most often due to defective renal excretion. It is characterized clinically by electrocardiographic abnormalities (elevated T waves and depressed P waves, and eventually by atrial asystole). In severe cases, weakness and flaccid paralysis may occur. (Dorland, 27th ed) Hyperpotassemia,Hyperkalemias,Hyperpotassemias
D000450 Aldosterone A hormone secreted by the ADRENAL CORTEX that regulates electrolyte and water balance by increasing the renal retention of sodium and the excretion of potassium. Aldosterone, (+-)-Isomer,Aldosterone, (11 beta,17 alpha)-Isomer

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