Causes, kinetics and clinical implications of post-hemodialysis urea rebound. 1988

L A Pedrini, and S Zereik, and S Rasmy
Servizio di Dialisi, Ospedale S Isidoro, Trescore Balneario, Bergamo, Italy.

The rapid increase in end-dialysis urea concentration (Co) immediately after the end of dialysis (HD), which greatly exceeds that expected as an effect of urea generation and defined as "net rebound," was assessed in 21 chronic HD patients. The curve of serial values of net rebound correlated (r = 0.70) with the theoretical curve predicted by the two pool urea kinetics model (UKM). A mean equilibrium concentration (Ce) was achieved in 48 minutes, with a 7.58% increase in Co. Stabilized rebound (Re) was compared after four different HD procedures, and significant correlations were found between the magnitude of Re and the indexes of HD efficiency, dialyzer clearance (r = 0.75) and Kt/V (r = 0.68). The highest values of Re (8.6% and 8.8%) were observed after the procedures with largest urea removal, irrespective of the biocompatibility conditions (new or reused dialyzers). The single pool UKM applied with the stabilized end-HD urea concentration Ce instead of Co resulted in more physiological values of urea distribution volume (56.1% vs. 50.5% of body wt) and in lower values of Kt/V (0.64 vs. 0.73, P less than 0.001) and protein catabolic rate (1.07 vs. 1.17 g/kg/day, P less than 0.001). A reequilibration process, rather than protein hypercatabolism, seems to be responsible for most rebound, the magnitude of which correlated with the efficiency of the procedure. Only by considering Ce as the true end-HD urea concentration it is possible to minimize the errors arising from the application of a single pool analysis to a two pool system.

UI MeSH Term Description Entries
D007700 Kinetics The rate dynamics in chemical or physical systems.
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D008954 Models, Biological Theoretical representations that simulate the behavior or activity of biological processes or diseases. For disease models in living animals, DISEASE MODELS, ANIMAL is available. Biological models include the use of mathematical equations, computers, and other electronic equipment. Biological Model,Biological Models,Model, Biological,Models, Biologic,Biologic Model,Biologic Models,Model, Biologic
D001825 Body Fluid Compartments The two types of spaces between which water and other body fluids are distributed: extracellular and intracellular. Body Fluid Compartment,Compartment, Body Fluid,Compartments, Body Fluid,Fluid Compartment, Body,Fluid Compartments, Body
D005260 Female Females
D006435 Renal Dialysis Therapy for the insufficient cleansing of the BLOOD by the kidneys based on dialysis and including hemodialysis, PERITONEAL DIALYSIS, and HEMODIAFILTRATION. Dialysis, Extracorporeal,Dialysis, Renal,Extracorporeal Dialysis,Hemodialysis,Dialyses, Extracorporeal,Dialyses, Renal,Extracorporeal Dialyses,Hemodialyses,Renal Dialyses
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D014508 Urea A compound formed in the liver from ammonia produced by the deamination of amino acids. It is the principal end product of protein catabolism and constitutes about one half of the total urinary solids. Basodexan,Carbamide,Carmol
D014511 Uremia A clinical syndrome associated with the retention of renal waste products or uremic toxins in the blood. It is usually the result of RENAL INSUFFICIENCY. Most uremic toxins are end products of protein or nitrogen CATABOLISM, such as UREA or CREATININE. Severe uremia can lead to multiple organ dysfunctions with a constellation of symptoms. Uremias

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