BIOMAT Database Logo BIOMAT Database Logo

Mast Cell Deficiency Protects Susceptible BALB/c Mice from Progressive Murine Cutaneous Leishmaniasis. 2021

Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Department of Dermatology, University Hospital of the Johannes Gutenberg-University Mainz, Mainz, Germany.

UI MeSH Term Description Entries
D008407 Mast Cells Granulated cells that are found in almost all tissues, most abundantly in the skin and the gastrointestinal tract. Like the BASOPHILS, mast cells contain large amounts of HISTAMINE and HEPARIN. Unlike basophils, mast cells normally remain in the tissues and do not circulate in the blood. Mast cells, derived from the bone marrow stem cells, are regulated by the STEM CELL FACTOR. Basophils, Tissue,Basophil, Tissue,Cell, Mast,Cells, Mast,Mast Cell,Tissue Basophil,Tissue Basophils
D008807 Mice, Inbred BALB C An inbred strain of mouse that is widely used in IMMUNOLOGY studies and cancer research. BALB C Mice, Inbred,BALB C Mouse, Inbred,Inbred BALB C Mice,Inbred BALB C Mouse,Mice, BALB C,Mouse, BALB C,Mouse, Inbred BALB C,BALB C Mice,BALB C Mouse
D008822 Mice, Transgenic Laboratory mice that have been produced from a genetically manipulated EGG or EMBRYO, MAMMALIAN. Transgenic Mice,Founder Mice, Transgenic,Mouse, Founder, Transgenic,Mouse, Transgenic,Mice, Transgenic Founder,Transgenic Founder Mice,Transgenic Mouse
D004195 Disease Models, Animal Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases. Animal Disease Model,Animal Disease Models,Disease Model, Animal
D004198 Disease Susceptibility A constitution or condition of the body which makes the tissues react in special ways to certain extrinsic stimuli and thus tends to make the individual more than usually susceptible to certain diseases. Diathesis,Susceptibility, Disease,Diatheses,Disease Susceptibilities,Susceptibilities, Disease
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000818 Animals Unicellular or multicellular, heterotrophic organisms, that have sensation and the power of voluntary movement. Under the older five kingdom paradigm, Animalia was one of the kingdoms. Under the modern three domain model, Animalia represents one of the many groups in the domain EUKARYOTA. Animal,Metazoa,Animalia
D016773 Leishmaniasis, Cutaneous An endemic disease that is characterized by the development of single or multiple localized lesions on exposed areas of skin that typically ulcerate. The disease has been divided into Old and New World forms. Old World leishmaniasis is separated into three distinct types according to epidemiology and clinical manifestations and is caused by species of the L. tropica and L. aethiopica complexes as well as by species of the L. major genus. New World leishmaniasis, also called American leishmaniasis, occurs in South and Central America and is caused by species of the L. mexicana or L. braziliensis complexes. Leishmaniasis, American,Leishmaniasis, New World,Leishmaniasis, Old World,Oriental Sore,American Leishmaniasis,Cutaneous Leishmaniases,Cutaneous Leishmaniasis,Leishmaniases, Cutaneous,New World Leishmaniasis,Old World Leishmaniasis,Sore, Oriental
D051379 Mice The common name for the genus Mus. Mice, House,Mus,Mus musculus,Mice, Laboratory,Mouse,Mouse, House,Mouse, Laboratory,Mouse, Swiss,Mus domesticus,Mus musculus domesticus,Swiss Mice,House Mice,House Mouse,Laboratory Mice,Laboratory Mouse,Mice, Swiss,Swiss Mouse,domesticus, Mus musculus
D058504 Th17 Cells A subset of helper-effector T-lymphocytes which synthesize and secrete INTERLEUKINS IL-17; IL-17F; and IL-22. These cytokines are involved in host defenses and tissue inflammation in autoimmune diseases. T Helper 17 Cell,TH-17 Cell,Th17 Cell,Type 17 Helper T Cell,T Helper 17 Cells,TH-17 Cells,Type 17 Helper T Cells,Cell, TH-17,Cell, Th17,Cells, TH-17,Cells, Th17,TH 17 Cell,TH 17 Cells

Related Publications

Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Pretreatment with recombinant Flt3 ligand partially protects against progressive cutaneous leishmaniasis in susceptible BALB/c mice.
February 2001, Infection and immunity,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Resistance to cutaneous leishmaniasis in genetically susceptible BALB/c mice.
October 1981, The Australian journal of experimental biology and medical science,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Cutaneous leishmaniasis. The defect in T cell influx in BALB/c mice.
February 1987, The Journal of experimental medicine,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Mast Cell Deficiency Protects Mice from Surgery-Induced Neuroinflammation.
January 2020, Mediators of inflammation,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Therapeutic effect of anti-L3T4 monoclonal antibody GK1.5 on cutaneous leishmaniasis in genetically-susceptible BALB/c mice.
September 1985, Journal of immunology (Baltimore, Md. : 1950),
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Vaccine-induced immunity against cutaneous leishmaniasis in BALB/c mice.
April 1988, Infection and immunity,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Mast cell-deficiency protects mice from streptozotocin-induced diabetic cardiomyopathy.
June 2019, Translational research : the journal of laboratory and clinical medicine,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
2,3,7,8-tetrachlorodibenzo-p-dioxin slows the progression of experimental cutaneous Leishmaniasis in susceptible BALB/c and SCID mice.
January 2013, PloS one,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Interleukin-4-independent acceleration of cutaneous leishmaniasis in susceptible BALB/c mice following treatment with anti-CTLA4 antibody.
December 1999, Infection and immunity,
Kirsten Dietze-Schwonberg, and Beate Lorenz, and Ann-Kathrin Hartmann, and Michael Stassen, and Esther von Stebut
Leishmania (Viannia) panamensis-induced cutaneous leishmaniasis in Balb/c mice: pathology.
October 1993, International journal of experimental pathology,
Copied contents to your clipboard!