| D003530 |
Cyprinidae |
A family of freshwater fish comprising the minnows or CARPS. |
Barbels,Chub,Dace,Minnows,Roach (Fish),Shiner,Tench,Tinca,Barbus,Rutilus rutilus,Tinca tinca,Chubs,Shiners,Tinca tincas,tinca, Tinca |
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| D005393 |
Fish Diseases |
Diseases of freshwater, marine, hatchery or aquarium fish. This term includes diseases of both teleosts (true fish) and elasmobranchs (sharks, rays and skates). |
Disease, Fish,Diseases, Fish,Fish Disease |
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| D006054 |
Goldfish |
Common name for Carassius auratus, a type of carp (CARPS). |
Carassius auratus |
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| D000818 |
Animals |
Unicellular or multicellular, heterotrophic organisms, that have sensation and the power of voluntary movement. Under the older five kingdom paradigm, Animalia was one of the kingdoms. Under the modern three domain model, Animalia represents one of the many groups in the domain EUKARYOTA. |
Animal,Metazoa,Animalia |
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| D000821 |
Animal Feed |
Foodstuff used especially for domestic and laboratory animals, or livestock. |
Fodder,Animal Feeds,Feed, Animal,Feeds, Animal,Fodders |
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| D000830 |
Animals, Laboratory |
Animals used or intended for use in research, testing, or teaching. |
Laboratory Animals,Animal, Laboratory,Laboratory Animal |
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| D012643 |
Selenium |
An element with the atomic symbol Se, atomic number 34, and atomic weight 78.97. It is an essential micronutrient for mammals and other animals but is toxic in large amounts. Selenium protects intracellular structures against oxidative damage. It is an essential component of GLUTATHIONE PEROXIDASE. |
Selenium-80,Selenium 80 |
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| D012758 |
Shellfish |
Aquatic invertebrates belonging to the phylum MOLLUSCA or the subphylum CRUSTACEA, and used as food. |
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| D014811 |
Vitamin E Deficiency |
A nutritional condition produced by a deficiency of VITAMIN E in the diet, characterized by posterior column and spinocerebellar tract abnormalities, areflexia, ophthalmoplegia, and disturbances of gait, proprioception, and vibration. In premature infants vitamin E deficiency is associated with hemolytic anemia, thrombocytosis, edema, intraventricular hemorrhage, and increasing risk of retrolental fibroplasia and bronchopulmonary dysplasia. An apparent inborn error of vitamin E metabolism, named familial isolated vitamin E deficiency, has recently been identified. (Cecil Textbook of Medicine, 19th ed, p1181) |
Deficiency, Vitamin E,Deficiencies, Vitamin E,Vitamin E Deficiencies |
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