Plasma testosterone in rats exposed to ethanol during vitamin E deficiency. 1987

J T Työppönen, and C J Eriksson
Research Laboratories of the Finnish State Alcohol Company (Alko Ltd.), Helsinki.

The effects of short and long-term ethanol administration on plasma testosterone level were studied in rats with vitamin E deficiency. The animals underwent a vitamin E depletion period of 5 weeks followed by an acute dose of ethanol (1 g/kg body wt i.p.). Two hours after the ethanol dose, the plasma testosterone level had decreased both in a control group (-32%, nonsignificant) and in the vitamin E deficient group (-45%, P less than 0.05) as compared with saline-treated rats. The rats which had received an ethanol dose were then exposed to ethanol in the drinking water (10% w/v) for two weeks, after which the acute ethanol dose was repeated. One hour after this second acute dose of ethanol, the plasma testosterone showed a decrease of 24% (nonsignificant) in the control group and 50% (P less than 0.05) in the vitamin E deficient group compared with saline-treated rats. Two hours after the second acute dose of ethanol, the plasma testosterone level was significantly (P less than 0.05) down both in the control group and in the vitamin E deficient group (-41 and -54%, respectively). Thus, vitamin E deficiency strengthened the effect of acute ethanol treatment on plasma testosterone in rats. Long-term exposure to ethanol appeared to sensitize rats to acute doses of ethanol, as judged by the fall in plasma testosterone levels. The present results are in agreement with previous findings in vitro thus supporting the notion that free radicals arising during ethanol oxidation have an inhibitory effect on testosterone synthesis.

UI MeSH Term Description Entries
D008297 Male Males
D000431 Ethanol A clear, colorless liquid rapidly absorbed from the gastrointestinal tract and distributed throughout the body. It has bactericidal activity and is used often as a topical disinfectant. It is widely used as a solvent and preservative in pharmaceutical preparations as well as serving as the primary ingredient in ALCOHOLIC BEVERAGES. Alcohol, Ethyl,Absolute Alcohol,Grain Alcohol,Alcohol, Absolute,Alcohol, Grain,Ethyl Alcohol
D000818 Animals Unicellular or multicellular, heterotrophic organisms, that have sensation and the power of voluntary movement. Under the older five kingdom paradigm, Animalia was one of the kingdoms. Under the modern three domain model, Animalia represents one of the many groups in the domain EUKARYOTA. Animal,Metazoa,Animalia
D013737 Testis The male gonad containing two functional parts: the SEMINIFEROUS TUBULES for the production and transport of male germ cells (SPERMATOGENESIS) and the interstitial compartment containing LEYDIG CELLS that produce ANDROGENS. Testicles,Testes,Testicle
D013739 Testosterone A potent androgenic steroid and major product secreted by the LEYDIG CELLS of the TESTIS. Its production is stimulated by LUTEINIZING HORMONE from the PITUITARY GLAND. In turn, testosterone exerts feedback control of the pituitary LH and FSH secretion. Depending on the tissues, testosterone can be further converted to DIHYDROTESTOSTERONE or ESTRADIOL. 17-beta-Hydroxy-4-Androsten-3-one,17-beta-Hydroxy-8 alpha-4-Androsten-3-one,8-Isotestosterone,AndroGel,Androderm,Andropatch,Androtop,Histerone,Sterotate,Sustanon,Testim,Testoderm,Testolin,Testopel,Testosterone Sulfate,17 beta Hydroxy 4 Androsten 3 one,17 beta Hydroxy 8 alpha 4 Androsten 3 one,8 Isotestosterone
D013997 Time Factors Elements of limited time intervals, contributing to particular results or situations. Time Series,Factor, Time,Time Factor
D014811 Vitamin E Deficiency A nutritional condition produced by a deficiency of VITAMIN E in the diet, characterized by posterior column and spinocerebellar tract abnormalities, areflexia, ophthalmoplegia, and disturbances of gait, proprioception, and vibration. In premature infants vitamin E deficiency is associated with hemolytic anemia, thrombocytosis, edema, intraventricular hemorrhage, and increasing risk of retrolental fibroplasia and bronchopulmonary dysplasia. An apparent inborn error of vitamin E metabolism, named familial isolated vitamin E deficiency, has recently been identified. (Cecil Textbook of Medicine, 19th ed, p1181) Deficiency, Vitamin E,Deficiencies, Vitamin E,Vitamin E Deficiencies
D051381 Rats The common name for the genus Rattus. Rattus,Rats, Laboratory,Rats, Norway,Rattus norvegicus,Laboratory Rat,Laboratory Rats,Norway Rat,Norway Rats,Rat,Rat, Laboratory,Rat, Norway,norvegicus, Rattus

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