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A determination was made of the renal tubular maximum reabsorptive capacity for phosphate (expressed as a function of the glomerular filtrate) in 33 patients with primary hypercalcaemic hyperparathyroidism and 30 control normal subjects. The difference between patients and controls for the maximum renal tubular reabsorptive capacity for phosphate (patients 0.526 +/- 0.16 mmol/l, controls 1.010 +/- 0.14 mmol/l) was highly significant (p less than 0.001). Successful removal of a single parathyroid adenoma in 19 patients was associated with a highly significant rise in the renal tubular maximum reabsorptive capacity for phosphate (1.005 +/- 0.21 mmol/l, p less than 0.001). We did not find any correlation between the renal tubular maximum reabsorptive capacity for phosphate and serum immunoreactive PTH level. Low renal tubular maximum reabsorptive capacity for phosphate in the presence of hypercalcaemia is highly suggestive of primary hyperparathyroidism.
P D Broulik, and
V Pacovský
October 1964,
Clinical science,
P D Broulik, and
V Pacovský
July 1980,
Clinical endocrinology,
P D Broulik, and
V Pacovský
March 1947,
Federation proceedings,
P D Broulik, and
V Pacovský
April 1964,
The Journal of clinical endocrinology and metabolism,
P D Broulik, and
V Pacovský
August 1971,
Lancet (London, England),
P D Broulik, and
V Pacovský
October 1977,
Die Medizinische Welt,
P D Broulik, and
V Pacovský
January 1983,
The American journal of physiology,
P D Broulik, and
V Pacovský
February 1967,
Nihon Hinyokika Gakkai zasshi. The japanese journal of urology,
P D Broulik, and
V Pacovský
October 1968,
Experientia,
P D Broulik, and
V Pacovský
May 2002,
World journal of surgery,
