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First mouse model of infant acute myeloid leukemia with t(7;12)(q36;p17).
2024
Zivojin Jevtic, and Juerg Schwaller
University Children Hospital Basel, Department of Biomedicine, University of Basel.
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Zivojin Jevtic, and Juerg Schwaller
Three-way complex translocations in infant acute myeloid leukemia with t(7;12)(q36;p13): the incidence and correlation of a HLXB9 overexpression.
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t(7;12)(q36;p13), a new recurrent translocation involving ETV6 in infant leukemia.
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Aberrant MNX1 expression associated with t(7;12)(q36;p13) pediatric acute myeloid leukemia induces the disease through altering histone methylation.
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t(7;12)(q36;p13) and t(7;12)(q32;p13)--translocations involving ETV6 in children 18 months of age or younger with myeloid disorders.
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Backtracking NOM1::ETV6 fusion to neonatal pathogenesis of t(7;12) (q36;p13) infant AML.
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Dicentric (7;12)(p11;p11) in T/Myeloid Mixed-Phenotype Acute Leukemia
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Paediatric acute myeloid leukaemia with the t(7;12)(q36;p13) rearrangement: a review of the biological and clinical management aspects.
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An induced pluripotent stem cell t(7;12)(q36;p13) acute myeloid leukemia model shows high expression of MNX1 and a block in differentiation of the erythroid and megakaryocytic lineages.
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