[An autopsy case of spontaneous bacterial peritonitis associated with fulminant type A hepatitis]. 1985

H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki

UI MeSH Term Description Entries
D008099 Liver A large lobed glandular organ in the abdomen of vertebrates that is responsible for detoxification, metabolism, synthesis and storage of various substances. Livers
D008297 Male Males
D010538 Peritonitis INFLAMMATION of the PERITONEUM lining the ABDOMINAL CAVITY as the result of infectious, autoimmune, or chemical processes. Primary peritonitis is due to infection of the PERITONEAL CAVITY via hematogenous or lymphatic spread and without intra-abdominal source. Secondary peritonitis arises from the ABDOMINAL CAVITY itself through RUPTURE or ABSCESS of intra-abdominal organs. Primary Peritonitis,Secondary Peritonitis,Peritonitis, Primary,Peritonitis, Secondary
D004926 Escherichia coli A species of gram-negative, facultatively anaerobic, rod-shaped bacteria (GRAM-NEGATIVE FACULTATIVELY ANAEROBIC RODS) commonly found in the lower part of the intestine of warm-blooded animals. It is usually nonpathogenic, but some strains are known to produce DIARRHEA and pyogenic infections. Pathogenic strains (virotypes) are classified by their specific pathogenic mechanisms such as toxins (ENTEROTOXIGENIC ESCHERICHIA COLI), etc. Alkalescens-Dispar Group,Bacillus coli,Bacterium coli,Bacterium coli commune,Diffusely Adherent Escherichia coli,E coli,EAggEC,Enteroaggregative Escherichia coli,Enterococcus coli,Diffusely Adherent E. coli,Enteroaggregative E. coli,Enteroinvasive E. coli,Enteroinvasive Escherichia coli
D006501 Hepatic Encephalopathy A syndrome characterized by central nervous system dysfunction in association with LIVER FAILURE, including portal-systemic shunts. Clinical features include lethargy and CONFUSION (frequently progressing to COMA); ASTERIXIS; NYSTAGMUS, PATHOLOGIC; brisk oculovestibular reflexes; decorticate and decerebrate posturing; MUSCLE SPASTICITY; and bilateral extensor plantar reflexes (see REFLEX, BABINSKI). ELECTROENCEPHALOGRAPHY may demonstrate triphasic waves. (From Adams et al., Principles of Neurology, 6th ed, pp1117-20; Plum & Posner, Diagnosis of Stupor and Coma, 3rd ed, p222-5) Encephalopathy, Hepatic,Portosystemic Encephalopathy,Encephalopathy, Hepatocerebral,Encephalopathy, Portal-Systemic,Encephalopathy, Portosystemic,Fulminant Hepatic Failure with Cerebral Edema,Hepatic Coma,Hepatic Stupor,Hepatocerebral Encephalopathy,Portal-Systemic Encephalopathy,Coma, Hepatic,Comas, Hepatic,Encephalopathies, Hepatic,Encephalopathies, Hepatocerebral,Encephalopathies, Portal-Systemic,Encephalopathies, Portosystemic,Encephalopathy, Portal Systemic,Hepatic Comas,Hepatic Encephalopathies,Hepatic Stupors,Hepatocerebral Encephalopathies,Portal Systemic Encephalopathy,Portal-Systemic Encephalopathies,Portosystemic Encephalopathies,Stupor, Hepatic,Stupors, Hepatic
D006506 Hepatitis A INFLAMMATION of the LIVER in humans caused by a member of the HEPATOVIRUS genus, HUMAN HEPATITIS A VIRUS. It can be transmitted through fecal contamination of food or water. Hepatitis, Infectious,Infectious Hepatitis,Hepatitides, Infectious,Infectious Hepatitides
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000328 Adult A person having attained full growth or maturity. Adults are of 19 through 44 years of age. For a person between 19 and 24 years of age, YOUNG ADULT is available. Adults
D012772 Shock, Septic Sepsis associated with HYPOTENSION or hypoperfusion despite adequate fluid resuscitation. Perfusion abnormalities may include but are not limited to LACTIC ACIDOSIS; OLIGURIA; or acute alteration in mental status. Endotoxin Shock,Septic Shock,Shock, Endotoxic,Shock, Toxic,Toxic Shock,Toxic Shock Syndrome,Endotoxin Shocks,Shock Syndrome, Toxic,Shock, Endotoxin,Shocks, Endotoxin,Toxic Shock Syndromes

Related Publications

H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
April 1982, Digestive diseases and sciences,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
April 1983, Digestive diseases and sciences,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
June 1982, Journal of clinical gastroenterology,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
January 1998, Journal of gastroenterology and hepatology,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
December 1998, Diabetes care,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
January 1981, The Kurume medical journal,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
February 1986, Journal of clinical gastroenterology,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
August 1981, Clinical nephrology,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
October 2001, Journal of clinical gastroenterology,
H Tsuji, and K Murai, and E Kajiwara, and K Akagi, and M Ikeda, and M Fujishima, and K Ayukawa, and T Iwaki
January 2000, The American journal of gastroenterology,
Copied contents to your clipboard!