BIOMAT Database Logo BIOMAT Database Logo

Ibuprofen may induce meningitis in (NZB X NZW)F1 mice. 1985

S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas

UI MeSH Term Description Entries
D007052 Ibuprofen A non-steroidal anti-inflammatory agent with analgesic, antipyretic, and anti-inflammatory properties Advil,Benzeneacetic Acid, alpha-methyl-4-(2-methylpropyl)- trimethylsilyl ester,Brufen,Ibumetin,Ibuprofen, (+-)-Isomer,Ibuprofen, (R)-Isomer,Ibuprofen, (S)-Isomer,Ibuprofen, Aluminum Salt,Ibuprofen, Calcium Salt,Ibuprofen, Copper (2+) Salt,Ibuprofen, Magnesium Salt,Ibuprofen, Potassium Salt,Ibuprofen, Sodium Salt,Ibuprofen, Zinc Salt,Ibuprofen-Zinc,Motrin,Nuprin,Rufen,Salprofen,Trauma-Dolgit Gel,alpha-Methyl-4-(2-methylpropyl)benzeneacetic Acid,Ibuprofen Zinc,Trauma Dolgit Gel
D008180 Lupus Erythematosus, Systemic A chronic, relapsing, inflammatory, and often febrile multisystemic disorder of connective tissue, characterized principally by involvement of the skin, joints, kidneys, and serosal membranes. It is of unknown etiology, but is thought to represent a failure of the regulatory mechanisms of the autoimmune system. The disease is marked by a wide range of system dysfunctions, an elevated erythrocyte sedimentation rate, and the formation of LE cells in the blood or bone marrow. Libman-Sacks Disease,Lupus Erythematosus Disseminatus,Systemic Lupus Erythematosus,Disease, Libman-Sacks,Libman Sacks Disease
D008581 Meningitis Inflammation of the coverings of the brain and/or spinal cord, which consist of the PIA MATER; ARACHNOID; and DURA MATER. Infections (viral, bacterial, and fungal) are the most common causes of this condition, but subarachnoid hemorrhage (HEMORRHAGES, SUBARACHNOID), chemical irritation (chemical MENINGITIS), granulomatous conditions, neoplastic conditions (CARCINOMATOUS MENINGITIS), and other inflammatory conditions may produce this syndrome. (From Joynt, Clinical Neurology, 1994, Ch24, p6) Pachymeningitis,Meningitides,Pachymeningitides
D008582 Meningitis, Aseptic A syndrome characterized by headache, neck stiffness, low grade fever, and CSF lymphocytic pleocytosis in the absence of an acute bacterial pathogen. Viral meningitis is the most frequent cause although MYCOPLASMA INFECTIONS; RICKETTSIA INFECTIONS; diagnostic or therapeutic procedures; NEOPLASTIC PROCESSES; septic perimeningeal foci; and other conditions may result in this syndrome. (From Adams et al., Principles of Neurology, 6th ed, p745) Aseptic Meningitis
D008807 Mice, Inbred BALB C An inbred strain of mouse that is widely used in IMMUNOLOGY studies and cancer research. BALB C Mice, Inbred,BALB C Mouse, Inbred,Inbred BALB C Mice,Inbred BALB C Mouse,Mice, BALB C,Mouse, BALB C,Mouse, Inbred BALB C,BALB C Mice,BALB C Mouse
D008814 Mice, Inbred NZB An inbred strain of mouse that is widely used as a model for AUTOIMMUNE DISEASES such as SYSTEMIC LUPUS ERYTHEMATOSUS. Mice, NZB,Mouse, Inbred NZB,Mouse, NZB,Inbred NZB Mice,Inbred NZB Mouse,NZB Mice,NZB Mice, Inbred,NZB Mouse,NZB Mouse, Inbred
D001921 Brain The part of CENTRAL NERVOUS SYSTEM that is contained within the skull (CRANIUM). Arising from the NEURAL TUBE, the embryonic brain is comprised of three major parts including PROSENCEPHALON (the forebrain); MESENCEPHALON (the midbrain); and RHOMBENCEPHALON (the hindbrain). The developed brain consists of CEREBRUM; CEREBELLUM; and other structures in the BRAIN STEM. Encephalon
D004195 Disease Models, Animal Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases. Animal Disease Model,Animal Disease Models,Disease Model, Animal
D005260 Female Females
D000818 Animals Unicellular or multicellular, heterotrophic organisms, that have sensation and the power of voluntary movement. Under the older five kingdom paradigm, Animalia was one of the kingdoms. Under the modern three domain model, Animalia represents one of the many groups in the domain EUKARYOTA. Animal,Metazoa,Animalia

Related Publications

S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Relative inability to induce tolerance in adult NZB and NZB-NZW F1 mice.
January 1969, The Journal of experimental medicine,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Interleukin-6 exacerbates glomerulonephritis in (NZB x NZW)F1 mice.
May 1994, The American journal of pathology,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Immunohistochemical characterization of sialadenitis in NZB X NZW F1 mice.
January 1987, Clinical immunology and immunopathology,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Monoclonal macroglobulinemia in NZB-NZW F1 mice.
October 1973, The Journal of experimental medicine,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Suppressor cells and immunodeficiency in (NZB x NZW)F1 hybrid mice.
June 1979, European journal of immunology,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Cyclophosphamide treatment of kidney disease in (NZB x NZW) F1 mice.
June 1966, Lancet (London, England),
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Intrinsic B cell defects in NZB and NZW mice contribute to systemic lupus erythematosus in (NZB x NZW)F1 mice.
September 1996, The Journal of experimental medicine,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Pathological changes of thymic epithelial cells and autoimmune disease in NZB, NZW and (NZB x NZW)F1 mice.
February 1967, Immunology,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
Differences between CD8+ T cells in lupus-prone (NZB x NZW) F1 mice and healthy (BALB/c x NZW) F1 mice may influence autoimmunity in the lupus model.
September 2004, European journal of immunology,
S Berliner, and A Weinberger, and Y Shoenfeld, and U Sandbank, and B Hazaz, and H Joshua, and M Rosen, and J Pinkhas
The contribution of NZW genes to lupus-like disease in (NZB x NZW)F1 mice.
May 1987, The Journal of experimental medicine,
Copied contents to your clipboard!