-transparent.png){kind=logo}
Histological and biochemical examinations of the adrenals and plasma of rats for 3 months exposed to hypokinesia have shown that low motor activity led to a decrease in blood corticosterone level in spite of adrenal cortex hypertrophy. The decreased corticosterone blood level was not indicative of adrenal exhaustion, as the adrenals produced a greater amount of corticosterone in response to additional stress stimulus (5-hour immobilisation of animals in an extended state), as compared to the control. The increased production of corticosterone in response to stress stimulus caused no structural transformations or delipoidization of the cortical substance. This indicated that the reserve potentials of the adrenals increased with the animal adaptation to hypokinesia. The major morphological indication of higher adrenal functional activity in hypokinetic animals was an enhanced destruction of lymphocytes in the thymus cortex, the target organ for corticosteroids produced by the adrenals in response to an additional stress stimulus.
A S Kaplanskiĭ, and
E V Vorotnikova
March 1974,
Voenno-meditsinskii zhurnal,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1986,
Kosmicheskaia biologiia i aviakosmicheskaia meditsina,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1983,
Kosmicheskaia biologiia i aviakosmicheskaia meditsina,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1975,
Agressologie: revue internationale de physio-biologie et de pharmacologie appliquees aux effets de l'agression,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1984,
Kosmicheskaia biologiia i aviakosmicheskaia meditsina,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1986,
Problemy endokrinologii,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1972,
Kosmicheskaia biologiia i meditsina,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1982,
Kosmicheskaia biologiia i aviakosmicheskaia meditsina,
A S Kaplanskiĭ, and
E V Vorotnikova
January 1980,
Neuroendocrinology,
A S Kaplanskiĭ, and
E V Vorotnikova
March 1974,
Endokrinologie,
