Biomaterial Database

Familial moniliasis, defective delayed hypersensitivity, and ACTH deficiency. 1972

J Neeman, and S Castells

UI	MeSH Term	Description	Entries
D006968	Hypersensitivity, Delayed	An increased reactivity to specific antigens mediated not by antibodies but by sensitized T CELLS.	Hypersensitivity, Tuberculin-Type,Hypersensitivity, Type IV,Tuberculin-Type Hypersensitivity,Type IV Hypersensitivity,Delayed Hypersensitivity,Delayed Hypersensitivities,Hypersensitivity, Tuberculin Type,Tuberculin Type Hypersensitivity,Tuberculin-Type Hypersensitivities,Type IV Hypersensitivities
D008797	Metyrapone	An inhibitor of the enzyme STEROID 11-BETA-MONOOXYGENASE. It is used as a test of the feedback hypothalamic-pituitary mechanism in the diagnosis of CUSHING SYNDROME.	Methbipyranone,Methopyrapone,Metopiron,Metopirone,Métopirone,SU 4885
D002179	Candidiasis, Cutaneous	Candidiasis of the skin manifested as eczema-like lesions of the interdigital spaces, perleche, or chronic paronychia. (Dorland, 27th ed)	Moniliasis, Cutaneous
D002648	Child	A person 6 to 12 years of age. An individual 2 to 5 years old is CHILD, PRESCHOOL.	Children
D006801	Humans	Members of the species Homo sapiens.	Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000224	Addison Disease	An adrenal disease characterized by the progressive destruction of the ADRENAL CORTEX, resulting in insufficient production of ALDOSTERONE and HYDROCORTISONE. Clinical symptoms include ANOREXIA; NAUSEA; WEIGHT LOSS; MUSCLE WEAKNESS; and HYPERPIGMENTATION of the SKIN due to increase in circulating levels of ACTH precursor hormone which stimulates MELANOCYTES.	Addison's Disease,Primary Adrenal Insufficiency,Primary Adrenocortical Insufficiency,Primary Hypoadrenalism,Addisons Disease,Adrenal Insufficiency, Primary,Adrenocortical Insufficiencies, Primary,Adrenocortical Insufficiency, Primary,Disease, Addison,Hypoadrenalism, Primary,Hypoadrenalisms, Primary,Insufficiencies, Primary Adrenocortical,Insufficiency, Primary Adrenocortical,Primary Adrenocortical Insufficiencies
D000324	Adrenocorticotropic Hormone	An anterior pituitary hormone that stimulates the ADRENAL CORTEX and its production of CORTICOSTEROIDS. ACTH is a 39-amino acid polypeptide of which the N-terminal 24-amino acid segment is identical in all species and contains the adrenocorticotrophic activity. Upon further tissue-specific processing, ACTH can yield ALPHA-MSH and corticotrophin-like intermediate lobe peptide (CLIP).	ACTH,Adrenocorticotropin,Corticotropin,1-39 ACTH,ACTH (1-39),Adrenocorticotrophic Hormone,Corticotrophin,Corticotrophin (1-39),Corticotropin (1-39),Hormone, Adrenocorticotrophic,Hormone, Adrenocorticotropic
D015068	17-Ketosteroids	Steroids that contain a ketone group at position 17.	17-Oxosteroids,17 Ketosteroids,17 Oxosteroids

Related Publications

J Neeman, and S Castells

Familial moniliasis, defective delayed hypersensitivity, and adrenocorticotropic hormone deficiency.

July 1971, The Journal of pediatrics,

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Chronic mucocutaneous moniliasis with impaired delayed hypersensitivity.

March 1970, Clinical and experimental immunology,

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"Acquired" agammaglobulinemia with defective delayed hypersensitivity.

August 1968, Annals of internal medicine,

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[ACTH receptor, ACTH receptor anomaly, and familial glucocorticoid deficiency].

July 1998, Nihon rinsho. Japanese journal of clinical medicine,

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Delayed hypersensitivity response and vitamin D deficiency.

January 1983, International journal for vitamin and nutrition research. Internationale Zeitschrift fur Vitamin- und Ernahrungsforschung. Journal international de vitaminologie et de nutrition,

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Defective lymphocyte transformation and delayed hypersensitivity in Wiskott-Aldrich syndrome.

April 1970, Journal of immunology (Baltimore, Md. : 1950),

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[FAMILIAL OCCURRENCE OF SUMMER-TYPE HYPERSENSITIVITY PNEUMONITIS IN DEFECTIVE HOME].

January 2018, Arerugi = [Allergy],

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The ability of ACTH and cortisone to alter delayed type bacterial hypersensitivity.

September 1950, Bulletin of the Johns Hopkins Hospital,

J Neeman, and S Castells

Congenital adrenal hypoplasia due to isolated familial ACTH deficiency.

January 1994, Pediatric pathology,