-transparent.png){kind=logo}
A 14-year-old boy with Goodpasture's syndrome induced by anti-glomerular-basement-membrane (gmb) antibody exhibited declining renal function, in association with a progressive increase in the level of serum anti-GBM antibody. Treatment with prednisone, cyclophosphamide, and plasmapheresis was associated with rapid disappearance of the serum anti-GMB antibody and temporary stabilization of renal function.
Related Publications
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
September 1983,
Archives of disease in childhood,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
January 1994,
Journal of clinical apheresis,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
March 1978,
Clinical nephrology,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
November 1979,
Mayo Clinic proceedings,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
July 1980,
Clinical pediatrics,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
May 1979,
Clinical and experimental immunology,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
May 1975,
British medical journal,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
June 1977,
The Medical journal of Australia,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
January 1977,
Verhandlungen der Deutschen Gesellschaft fur Innere Medizin,
R A Finch, and
E A Rutsky, and
E McGowan, and
C B Wilson
April 1976,
Lancet (London, England),
