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Under physiologic conditions the tcPO2 reflects the PO2 in the carotid artery. It is therefore a measure of the PO2 in the arteries supplying the brain. The response of the tcPO2 to fetal hypoxemia, as during the recovery period following hypoxemia, is delayed. The delayed response of the tcPO2 during recovery is generated by the release of catecholamines into the fetal circulation. The reading of the tcPO2 corresponds better with the course of the heart rate decelerations and the increased blood pressure than with the course of the PO2 in the fetal aorta. The tcPO2 is, in this context, a reliable measure for the peripheral oxygenation and vasoconstriction. The tcPO2 however should be applied with great care for studying the pathomechanism of fetal heart rate decelerations during labor.
W Künzel, and
E Kastendieck, and
C S Kurz
November 1980,
American journal of obstetrics and gynecology,
W Künzel, and
E Kastendieck, and
C S Kurz
April 1983,
Acta anaesthesiologica Scandinavica,
W Künzel, and
E Kastendieck, and
C S Kurz
October 1979,
The Journal of physiology,
W Künzel, and
E Kastendieck, and
C S Kurz
December 1971,
American journal of obstetrics and gynecology,
W Künzel, and
E Kastendieck, and
C S Kurz
December 1979,
Lancet (London, England),
W Künzel, and
E Kastendieck, and
C S Kurz
May 2002,
The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians,
W Künzel, and
E Kastendieck, and
C S Kurz
October 1981,
Critical care medicine,
W Künzel, and
E Kastendieck, and
C S Kurz
September 1961,
Journal of applied physiology,
W Künzel, and
E Kastendieck, and
C S Kurz
January 1976,
European journal of intensive care medicine,
W Künzel, and
E Kastendieck, and
C S Kurz
August 1967,
Diseases of the chest,
