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The time factor in the oxygen transfer from mother to fetus was studied by the tcPO2 technique. When a fairly constant fetal circulation made estimation possible, the decrease in fetal tcPO2 synchronous with and due to the uterine contractions in more than 400 events was always delayed 50--60 sec. Since the time lag in the increase in fetal tcPO2 following administration of pure oxygen to the mother was found to be of the same order of magnitude, the placental part of the delay in the oxygen transfer from mother to fetus can be assumed to be 50--60 sec. This determination supports the prevalent hypothesis that type II dips or late decelerations in the fetal heart rate are caused by hypoxia while type I dips or early decelerations are initiated by other factors.
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
September 1967,
American journal of obstetrics and gynecology,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
January 1964,
Bibliotheca gynaecologica,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
July 1982,
Anesthesia and analgesia,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
January 1960,
Journal of the American Medical Association,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
February 1969,
Archives of disease in childhood,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
January 1977,
Lancet (London, England),
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
July 1977,
The New England journal of medicine,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
January 1977,
Seminars in perinatology,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
August 1964,
Harefuah,
O Fall, and
B Ek, and
B A Nilsson, and
G Rooth
January 1955,
Archivio italiano di scienze farmacologiche,
