BIOMAT Database Logo BIOMAT Database Logo

Decreased erythrocyte glutathione peroxidase activity in neuronal lipofuscinosis (NCL) - corrected with selenium supplementation. 1977

T Westermarck, and M Sandholm

UI MeSH Term Description Entries
D008062 Lipofuscin A naturally occurring lipid pigment with histochemical characteristics similar to ceroid. It accumulates in various normal tissues and apparently increases in quantity with age.
D008064 Lipidoses Conditions characterized by abnormal lipid deposition due to disturbance in lipid metabolism, such as hereditary diseases involving lysosomal enzymes required for lipid breakdown. They are classified either by the enzyme defect or by the type of lipid involved. Lipidosis,Lipoidosis
D010544 Peroxidases Ovoperoxidase
D010860 Pigments, Biological Any normal or abnormal coloring matter in PLANTS; ANIMALS or micro-organisms. Biological Pigments
D002566 Ceroid A naturally occurring lipid pigment with histochemical characteristics similar to lipofuscin. It accumulates in various tissues in certain experimental and pathological conditions.
D004912 Erythrocytes Red blood cells. Mature erythrocytes are non-nucleated, biconcave disks containing HEMOGLOBIN whose function is to transport OXYGEN. Blood Cells, Red,Blood Corpuscles, Red,Red Blood Cells,Red Blood Corpuscles,Blood Cell, Red,Blood Corpuscle, Red,Erythrocyte,Red Blood Cell,Red Blood Corpuscle
D005979 Glutathione Peroxidase An enzyme catalyzing the oxidation of 2 moles of GLUTATHIONE in the presence of HYDROGEN PEROXIDE to yield oxidized glutathione and water. Cytosolic Glutathione Peroxidase,Glutathione Lipoperoxidase,Selenoglutathione Peroxidase,Glutathione Peroxidase, Cytosolic,Lipoperoxidase, Glutathione,Peroxidase, Glutathione,Peroxidase, Selenoglutathione
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D012643 Selenium An element with the atomic symbol Se, atomic number 34, and atomic weight 78.97. It is an essential micronutrient for mammals and other animals but is toxic in large amounts. Selenium protects intracellular structures against oxidative damage. It is an essential component of GLUTATHIONE PEROXIDASE. Selenium-80,Selenium 80

Related Publications

T Westermarck, and M Sandholm
Increased plasma and erythrocyte selenium concentrations but decreased erythrocyte glutathione peroxidase activity after selenium supplementation in children with Down syndrome.
November 1989, Acta paediatrica Scandinavica,
T Westermarck, and M Sandholm
Decreased erythrocyte glutathione peroxidase activity in multiple sclerosis.
January 1979, Acta neurologica Scandinavica,
T Westermarck, and M Sandholm
Selenium supplementation in X-linked muscular dystrophy. Effects on erythrocyte and serum selenium and on erythrocyte glutathione peroxidase activity.
November 1985, Acta paediatrica Scandinavica,
T Westermarck, and M Sandholm
Erythrocyte selenium-glutathione peroxidase activity is lower in patients with coronary atherosclerosis.
November 1997, Japanese heart journal,
T Westermarck, and M Sandholm
Selenium supplementation of Chinese women with habitually low selenium intake increases plasma selenium, plasma glutathione peroxidase activity, and milk selenium, but not milk glutathione peroxidase activity.
June 2000, The Journal of nutritional biochemistry,
T Westermarck, and M Sandholm
[Neuronal ceroid lipofuscinosis (NCL, Batten's disease)].
January 2000, Ryoikibetsu shokogun shirizu,
T Westermarck, and M Sandholm
Decreased Activity of Erythrocyte Catalase and Glutathione Peroxidase in Patients with Schizophrenia.
October 2022, Medicina (Kaunas, Lithuania),
T Westermarck, and M Sandholm
Increase in glutathione peroxidase activity in malaria parasite after selenium supplementation.
January 1996, Free radical biology & medicine,
T Westermarck, and M Sandholm
Plasma glutathione peroxidase after selenium supplementation in patients with reduced selenium state.
March 1982, European journal of pediatrics,
T Westermarck, and M Sandholm
Axonal enlargements (meganeurites) in neuronal ceroid lipofuscinosis (NCL).
January 1982, Ultrastructural pathology,
Copied contents to your clipboard!