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A patient with adrenocortical carcinoma with Cushing's syndrome is presented. This case seems to be most characterized by its very unusual urinary 17-ketosteroid (KS) fractionation, since markedly increased etiocholanolone was not accompanied by increments of either DHEA or androsterone. Determination of the plasma adrenocorticosteroids of the patient revealed normal DHEA and DHEA-S levels, moderately increased 17-OH-pregnenolone, and markedly increased 11-deoxycortisol. Therefore, it seems plausible that the unique 17-KS fractionation of this patient would have ensued as a result of remarkably decreased C17-20 lyase activity sufficient to nullify the increased 17-OH-pregnenolone and that markedly increased etiocholanolone might have been converted from 11-deoxycortisol.
H Watanobe, and
K Kannari, and
S Murabayashi, and
K Kimura, and
K Takebe, and
M Ojima, and
K Suzuki
April 1957,
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H Watanobe, and
K Kannari, and
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K Kimura, and
K Takebe, and
M Ojima, and
K Suzuki
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H Watanobe, and
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H Watanobe, and
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H Watanobe, and
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Nihon Naibunpi Gakkai zasshi,
H Watanobe, and
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K Suzuki
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H Watanobe, and
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H Watanobe, and
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K Takebe, and
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H Watanobe, and
K Kannari, and
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M Ojima, and
K Suzuki
September 1980,
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