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Studies during the last few years have revealed evidence of active secretion of HCO3(-) in the gastric mucosa both in vitro and in vivo. This transport process presumably has considerable importance for the protection of the gastric epithelium, disturbance must be considered a causal factor in the pathogenesis of gastric ulcer. The HCO3(-)-transport is subject to autonomic control. It is inhibited by drugs with an ulcerogenic action and stimulated by anti-ulcerogenic prostaglandins. The results available up to now raise the probability of the physiologic and pathophysiologic significance of the HCO3(-)-transport system. Clinical investigations must demonstrate the therapeutic efficacy of control of the alkali secretion in the gastric mucous membrane.
B Simon, and
P Müller, and
H Kather
July 1981,
Zhonghua nei ke za zhi,
B Simon, and
P Müller, and
H Kather
January 1980,
Gastroenterologie clinique et biologique,
B Simon, and
P Müller, and
H Kather
October 1974,
Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis,
B Simon, and
P Müller, and
H Kather
February 1976,
Klinische Wochenschrift,
B Simon, and
P Müller, and
H Kather
May 1981,
Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis,
B Simon, and
P Müller, and
H Kather
December 1978,
Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis,
B Simon, and
P Müller, and
H Kather
October 1979,
MMW, Munchener medizinische Wochenschrift,
B Simon, and
P Müller, and
H Kather
November 1977,
Therapeutische Umschau. Revue therapeutique,
B Simon, and
P Müller, and
H Kather
October 1980,
Nihon Shokakibyo Gakkai zasshi = The Japanese journal of gastro-enterology,
B Simon, and
P Müller, and
H Kather
November 1975,
Revista medica de Chile,
