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A 60-year-old woman, free of other disease, with severe clinical hyperthyroidism had normal total serum T4 with elevated free T4 concentration and T3 resin uptake characteristic of hyperthyroidism, with thyroid binding globulin (TBG) deficiency. However, serum TBG concentration as determined by immunoassay was normal, suggesting that the biochemical defect affecting the T4 binding sites did not affect is antigenicity. Her total and free serum T3 levels were low-normal with a markedly elevated reverse T3 concentration. These findings, in the presence of clinical hyperthyroidism, are consistent with an impairment in peripheral conversion of T4 to T3, apparently, in this case, due only to long-standing severe hyperthyroidism.
J F Caro, and
N G Sloane, and
J A Glennon
October 1975,
Australian and New Zealand journal of medicine,
J F Caro, and
N G Sloane, and
J A Glennon
January 1979,
Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme,
J F Caro, and
N G Sloane, and
J A Glennon
November 1978,
Revista clinica espanola,
J F Caro, and
N G Sloane, and
J A Glennon
May 1979,
Lancet (London, England),
J F Caro, and
N G Sloane, and
J A Glennon
June 1979,
Lancet (London, England),
J F Caro, and
N G Sloane, and
J A Glennon
June 1979,
Lancet (London, England),
J F Caro, and
N G Sloane, and
J A Glennon
September 1985,
Human nutrition. Clinical nutrition,
J F Caro, and
N G Sloane, and
J A Glennon
March 1980,
Acta endocrinologica,
J F Caro, and
N G Sloane, and
J A Glennon
January 1978,
Acta medica Scandinavica,
J F Caro, and
N G Sloane, and
J A Glennon
August 1974,
Clinical science and molecular medicine,
