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Corneal vascularization is attributable to the swelling and increased lactate concentration in the cornea, which are caused by damage to the oxidizing metabolism. Invasion of leukocytes stimulated by prostaglandins also promotes the increase in the lactate concentration. The following causal treatment is possible: 40% glucose to combat the swelling, 3% cystein to improve the oxidizing metabolism and counteract the increased lactate concentration, and 0.5% indomethacin suspension to prevent the formation of prostaglandins and hence the invasion of leukozytes.
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