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In order to achieve a hyperthyroid state, rats were treated for 7 days with thyroxine (150 microgram/100 g BW) or triiodothyronine (10 microgram/100 g BW). This regimen resulted in an enhanced activity of the microsomal ethanol oxidizing system. In addition, a decrease of hepatic alcohol dehydrogenase activity was observed under these experimental conditions, whereas hepatic catalase activity remained unchanged. These findings suggest that if chronic ethanol consumption simulates a functional "hyperthyroid hepatic state", increased rates of ethanol metabolism observed following prolonged alcohol intake might therefore be attributed at least in part to an induced activity of the microsomal ethanol oxidizing system in the liver.
F Moreno, and
M Minzlaff, and
K H Hauptmeier, and
R Teschke
August 1979,
Biochemical and biophysical research communications,
F Moreno, and
M Minzlaff, and
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R Teschke
January 1987,
General pharmacology,
F Moreno, and
M Minzlaff, and
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R Teschke
January 1982,
Enzyme,
F Moreno, and
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K H Hauptmeier, and
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F Moreno, and
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Biochemical pharmacology,
F Moreno, and
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K H Hauptmeier, and
R Teschke
January 2015,
Journal of investigative surgery : the official journal of the Academy of Surgical Research,
F Moreno, and
M Minzlaff, and
K H Hauptmeier, and
R Teschke
January 1988,
Comparative biochemistry and physiology. C, Comparative pharmacology and toxicology,
F Moreno, and
M Minzlaff, and
K H Hauptmeier, and
R Teschke
September 1979,
Journal of steroid biochemistry,
F Moreno, and
M Minzlaff, and
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R Teschke
February 1986,
Biochemical pharmacology,
F Moreno, and
M Minzlaff, and
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May 2002,
Experimental & molecular medicine,
