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A mutant defective in the induced excision of Tn10 was isolated by decreased papillation on MacConkey-galactose plates with mitomycin C. The mutation involved was characterized as recN by genetic mapping and complementation. This mutant, as well as a previously characterized recN mutant (recN262), showed a markedly decreased frequency of excision of Tn10 after treatment with UV or mitomycin C. These observations indicate that recN is involved in the induced excision of Tn10.
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