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Serial positron emission tomography with fludeoxyglucose F 18 in Creutzfeldt-Jakob disease. 1995
T Ogawa, and
A Inugami, and
H Fujita, and
J Hatazawa, and
E Shimosegawa, and
I Kanno, and
T Okudera, and
K Uemura, and
K Nagata
Department of Radiology and Nuclear Medicine, Research Institute of Brain and Blood Vessels-Akita, Japan.
We serially examined a patient with Creutzfeldt-Jakob disease using fludeoxyglucose F 18 and positron emission tomography. Marked cerebral hypometabolism reflected clinical deterioration in the early stages of Creutzfeldt-Jakob disease when no parenchymal abnormalities were present on MR imaging.
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