-transparent.png){kind=logo}
Seeds of barley, Hordeum vulgare L., with or without residual mercury were exposed to concentrations of ethyl methanesulfonate (EMS), maleic hydrazide (MH), methyl mercuric chloride (MMCl) and mercury-contaminated soil. Subsequently the endpoints measured were germination, seedling height, mitotic index, mitotic chromosome or spindle aberrations in embryonic shoot cells and meiotic chromosome aberration in pollen mother cells. The results unequivocally demonstrated that the seed-residual mercury conferred protection against the genotoxicity of EMS, MH, MMCl as well as mercury-contaminated soil in barley. The genotoxic adaptation to MH and MMCl was significantly prevented by pre-exposing the Hg-seeds to buthionine sulfoximine, an inhibitor of phytochelatin synthesis. Furthermore, compared to normal seedlings, the seedlings grown from Hg-seeds exhibited a higher amount of non-protein SH. The findings indicated a possible involvement of phytochelatins in the mercury-induced adaptive response.
A V Subhadra, and
K K Panda, and
B B Panda
February 2000,
Mutation research,
A V Subhadra, and
K K Panda, and
B B Panda
March 2005,
Mutation research,
A V Subhadra, and
K K Panda, and
B B Panda
May 2002,
Functional & integrative genomics,
A V Subhadra, and
K K Panda, and
B B Panda
January 2015,
Frontiers in plant science,
A V Subhadra, and
K K Panda, and
B B Panda
October 1974,
Plant physiology,
A V Subhadra, and
K K Panda, and
B B Panda
May 1976,
Plant physiology,
A V Subhadra, and
K K Panda, and
B B Panda
July 2015,
Genetika,
A V Subhadra, and
K K Panda, and
B B Panda
October 2017,
International journal of environmental research and public health,
A V Subhadra, and
K K Panda, and
B B Panda
January 2016,
Frontiers in plant science,
A V Subhadra, and
K K Panda, and
B B Panda
March 2023,
Scientific reports,
