BIOMAT Database Logo BIOMAT Database Logo

Carotid artery wall shear stress after LDL-apheresis in familial hypercholesterolemia. 1994

M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
Institute of Patologia Medica, University of Trieste.

UI MeSH Term Description Entries
D008077 Lipoproteins, LDL A class of lipoproteins of small size (18-25 nm) and light (1.019-1.063 g/ml) particles with a core composed mainly of CHOLESTEROL ESTERS and smaller amounts of TRIGLYCERIDES. The surface monolayer consists mostly of PHOSPHOLIPIDS, a single copy of APOLIPOPROTEIN B-100, and free cholesterol molecules. The main LDL function is to transport cholesterol and cholesterol esters to extrahepatic tissues. Low-Density Lipoprotein,Low-Density Lipoproteins,beta-Lipoprotein,beta-Lipoproteins,LDL(1),LDL(2),LDL-1,LDL-2,LDL1,LDL2,Low-Density Lipoprotein 1,Low-Density Lipoprotein 2,LDL Lipoproteins,Lipoprotein, Low-Density,Lipoproteins, Low-Density,Low Density Lipoprotein,Low Density Lipoprotein 1,Low Density Lipoprotein 2,Low Density Lipoproteins,beta Lipoprotein,beta Lipoproteins
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D001781 Blood Component Removal Any procedure in which blood is withdrawn from a donor, a portion is separated and retained, at the same time the remainder is returned to the donor. Apheresis,Pheresis,Aphereses,Blood Component Removals,Phereses,Removal, Blood Component
D001809 Blood Viscosity The internal resistance of the BLOOD to shear forces. The in vitro measure of whole blood viscosity is of limited clinical utility because it bears little relationship to the actual viscosity within the circulation, but an increase in the viscosity of circulating blood can contribute to morbidity in patients suffering from disorders such as SICKLE CELL ANEMIA and POLYCYTHEMIA. Blood Viscosities,Viscosities, Blood,Viscosity, Blood
D002339 Carotid Arteries Either of the two principal arteries on both sides of the neck that supply blood to the head and neck; each divides into two branches, the internal carotid artery and the external carotid artery. Arteries, Carotid,Artery, Carotid,Carotid Artery
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D006938 Hyperlipoproteinemia Type II A group of familial disorders characterized by elevated circulating cholesterol contained in either LOW-DENSITY LIPOPROTEINS alone or also in VERY-LOW-DENSITY LIPOPROTEINS (pre-beta lipoproteins). Hyperbetalipoproteinemia,Hypercholesterolemia, Essential,Hypercholesterolemia, Familial,Apolipoprotein B-100, Familial Defective,Apolipoprotein B-100, Familial Ligand-Defective,Familial Combined Hyperlipoproteinemia,Hyper-Low Density Lipoproteinemia,Hyper-Low-Density-Lipoproteinemia,Hyper-beta-Lipoproteinemia,Hypercholesterolemia, Autosomal Dominant,Hypercholesterolemia, Autosomal Dominant, Type B,Hypercholesterolemic Xanthomatosis, Familial,Hyperlipoproteinemia Type 2,Hyperlipoproteinemia Type IIa,Hyperlipoproteinemia Type IIb,Hyperlipoproteinemia, Type II,Hyperlipoproteinemia, Type IIa,LDL Receptor Disorder,Apolipoprotein B 100, Familial Defective,Apolipoprotein B 100, Familial Ligand Defective,Autosomal Dominant Hypercholesterolemia,Autosomal Dominant Hypercholesterolemias,Combined Hyperlipoproteinemia, Familial,Combined Hyperlipoproteinemias, Familial,Density Lipoproteinemia, Hyper-Low,Density Lipoproteinemias, Hyper-Low,Disorder, LDL Receptor,Disorders, LDL Receptor,Dominant Hypercholesterolemia, Autosomal,Dominant Hypercholesterolemias, Autosomal,Essential Hypercholesterolemia,Essential Hypercholesterolemias,Familial Combined Hyperlipoproteinemias,Familial Hypercholesterolemia,Familial Hypercholesterolemias,Familial Hypercholesterolemic Xanthomatoses,Familial Hypercholesterolemic Xanthomatosis,Hyper Low Density Lipoproteinemia,Hyper beta Lipoproteinemia,Hyper-Low Density Lipoproteinemias,Hyper-Low-Density-Lipoproteinemias,Hyper-beta-Lipoproteinemias,Hyperbetalipoproteinemias,Hypercholesterolemias, Autosomal Dominant,Hypercholesterolemias, Essential,Hypercholesterolemias, Familial,Hypercholesterolemic Xanthomatoses, Familial,Hyperlipoproteinemia Type 2s,Hyperlipoproteinemia Type IIas,Hyperlipoproteinemia Type IIbs,Hyperlipoproteinemia Type IIs,Hyperlipoproteinemia, Familial Combined,Hyperlipoproteinemias, Familial Combined,Hyperlipoproteinemias, Type II,Hyperlipoproteinemias, Type IIa,LDL Receptor Disorders,Lipoproteinemia, Hyper-Low Density,Lipoproteinemias, Hyper-Low Density,Receptor Disorder, LDL,Receptor Disorders, LDL,Type 2, Hyperlipoproteinemia,Type II Hyperlipoproteinemia,Type II Hyperlipoproteinemias,Type IIa Hyperlipoproteinemia,Type IIa Hyperlipoproteinemias,Xanthomatoses, Familial Hypercholesterolemic,Xanthomatosis, Familial Hypercholesterolemic
D000368 Aged A person 65 years of age or older. For a person older than 79 years, AGED, 80 AND OVER is available. Elderly

Related Publications

M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL-apheresis in the treatment familial hypercholesterolemia].
November 2014, Vnitrni lekarstvi,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
Myeloperoxidase modulation by LDL apheresis in familial hypercholesterolemia.
October 2011, Lipids in health and disease,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL apheresis in the treatment of familial hypercholesterolemia].
October 2014, Turk Kardiyoloji Dernegi arsivi : Turk Kardiyoloji Derneginin yayin organidir,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL-apheresis in the treatment of familial hypercholesterolemia].
January 1985, La Ricerca in clinica e in laboratorio,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
Shear stress-dependent platelet function after LDL cholesterol apheresis.
January 2004, Thrombosis research,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL-apheresis in patients with familial autosomal recessive hypercholesterolemia].
January 2012, Giornale italiano di nefrologia : organo ufficiale della Societa italiana di nefrologia,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
Pregnancy in homozygous familial hypercholesterolemia--Importance of LDL-apheresis.
May 2015, Atherosclerosis. Supplements,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL oxidation in homozygous familial hypercholesterolemia: effects of selective LDL-apheresis treatment].
May 1996, Cardiologia (Rome, Italy),
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
LDL-apheresis in homozygous LDL-receptor-defective familial hypercholesterolemia: the Munich experience.
December 2009, Atherosclerosis. Supplements,
M Bardelli, and F Cominotto, and M Fazio, and D Ussi, and P Bordin, and A Petrucco, and B Fabris, and F Fischetti, and R Lapasin, and R Carretta
[LDL apheresis--an effective treatment of homozygous familial hypercholesterolemia].
January 1990, Duodecim; laaketieteellinen aikakauskirja,
Copied contents to your clipboard!