BIOMAT Database Logo BIOMAT Database Logo

[Lipid metabolism and blood coagulation system function in patients with the late sequelae of closed craniocerebral trauma]. 1994

V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh

Long-term follow-up of the subjects who have suffered closed brain injury showed that in comparison to matched controls the injured young subjects demonstrated abnormalities in lipid metabolism and coagulation tending to atherosclerosis. The limits of the values by which the patients significantly differed from the controls as well as 7 specific for the patients signs of lipid metabolism and coagulation impairment have been determined. From 1 to 5 specific indications registered in 67 +/- 5% patients allowed the diagnosis in them of the injury sequelae. The mechanisms of the above atherosclerotic shifts may work through neuroregulatory derangements common for brain injury and atherosclerotic encephalopathy and enhancement of atherosclerosis course by the craniocerebral damage.

UI MeSH Term Description Entries
D008055 Lipids A generic term for fats and lipoids, the alcohol-ether-soluble constituents of protoplasm, which are insoluble in water. They comprise the fats, fatty oils, essential oils, waxes, phospholipids, glycolipids, sulfolipids, aminolipids, chromolipids (lipochromes), and fatty acids. (Grant & Hackh's Chemical Dictionary, 5th ed) Lipid
D008074 Lipoproteins Lipid-protein complexes involved in the transportation and metabolism of lipids in the body. They are spherical particles consisting of a hydrophobic core of TRIGLYCERIDES and CHOLESTEROL ESTERS surrounded by a layer of hydrophilic free CHOLESTEROL; PHOSPHOLIPIDS; and APOLIPOPROTEINS. Lipoproteins are classified by their varying buoyant density and sizes. Circulating Lipoproteins,Lipoprotein,Lipoproteins, Circulating
D008297 Male Males
D001777 Blood Coagulation The process of the interaction of BLOOD COAGULATION FACTORS that results in an insoluble FIBRIN clot. Blood Clotting,Coagulation, Blood,Blood Clottings,Clotting, Blood
D001780 Blood Coagulation Tests Laboratory tests for evaluating the individual's clotting mechanism. Coagulation Tests, Blood,Tests, Blood Coagulation,Blood Coagulation Test,Coagulation Test, Blood,Test, Blood Coagulation
D001930 Brain Injuries Acute and chronic (see also BRAIN INJURIES, CHRONIC) injuries to the brain, including the cerebral hemispheres, CEREBELLUM, and BRAIN STEM. Clinical manifestations depend on the nature of injury. Diffuse trauma to the brain is frequently associated with DIFFUSE AXONAL INJURY or COMA, POST-TRAUMATIC. Localized injuries may be associated with NEUROBEHAVIORAL MANIFESTATIONS; HEMIPARESIS, or other focal neurologic deficits. Brain Lacerations,Acute Brain Injuries,Brain Injuries, Acute,Brain Injuries, Focal,Focal Brain Injuries,Injuries, Acute Brain,Injuries, Brain,Acute Brain Injury,Brain Injury,Brain Injury, Acute,Brain Injury, Focal,Brain Laceration,Focal Brain Injury,Injuries, Focal Brain,Injury, Acute Brain,Injury, Brain,Injury, Focal Brain,Laceration, Brain,Lacerations, Brain
D002537 Intracranial Arteriosclerosis Vascular diseases characterized by thickening and hardening of the walls of ARTERIES inside the SKULL. There are three subtypes: (1) atherosclerosis with fatty deposits in the ARTERIAL INTIMA; (2) Monckeberg's sclerosis with calcium deposits in the media and (3) arteriolosclerosis involving the small caliber arteries. Clinical signs include HEADACHE; CONFUSION; transient blindness (AMAUROSIS FUGAX); speech impairment; and HEMIPARESIS. Cerebral Arteriosclerosis,Intracranial Atherosclerosis,Cerebral Atherosclerosis,Arterioscleroses, Cerebral,Arterioscleroses, Intracranial,Arteriosclerosis, Cerebral,Arteriosclerosis, Intracranial,Atheroscleroses, Cerebral,Atheroscleroses, Intracranial,Atherosclerosis, Cerebral,Atherosclerosis, Intracranial,Cerebral Arterioscleroses,Cerebral Atheroscleroses,Intracranial Arterioscleroses,Intracranial Atheroscleroses
D002908 Chronic Disease Diseases which have one or more of the following characteristics: they are permanent, leave residual disability, are caused by nonreversible pathological alteration, require special training of the patient for rehabilitation, or may be expected to require a long period of supervision, observation, or care (Dictionary of Health Services Management, 2d ed). For epidemiological studies chronic disease often includes HEART DISEASES; STROKE; CANCER; and diabetes (DIABETES MELLITUS, TYPE 2). Chronic Condition,Chronic Illness,Chronically Ill,Chronic Conditions,Chronic Diseases,Chronic Illnesses,Condition, Chronic,Disease, Chronic,Illness, Chronic
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man

Related Publications

V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[The prognosis of the immediate and late sequelae of mild closed craniocerebral trauma].
January 1998, Klinichna khirurhiia,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
Late sequelae following closed craniocerebral injuries
April 1961, Izvestiia na Mikrobiologicheskiia institut,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[Peripheral nervous system function in the late period of closed craniocerebral trauma in Afghanistan veterans].
January 1998, Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[Alkaline blood serum proteins in patients with the initial manifestations of cerebral arteriosclerosis and the late sequelae of mild closed craniocerebral trauma (comparative data)].
January 1995, Likars'ka sprava,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[Blood coagulation system and lipid metabolism in patients with arteriosclerosis].
November 1971, Vrachebnoe delo,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[The sequelae of a mild closed craniocerebral trauma and the prognosis of its late outcomes in patients with a unfavorable preinjury background].
January 1997, Likars'ka sprava,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[The validation of rational therapy in the sequelae of closed craniocerebral trauma].
January 1994, Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[Clinical signs and diagnosis of sequelae of mild closed craniocerebral trauma].
January 1998, Klinichna khirurhiia,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
[The significance of altered consciousness for late sequelae in craniocerebral trauma].
March 1987, Therapeutische Umschau. Revue therapeutique,
V I Taĭtslin, and I I Shogam, and V S Genes, and M K Azar'iants, and I A Zalogina, and L I Sedykh
Disseminated intravascular coagulation in patients with craniocerebral trauma.
December 1985, Journal of the Mississippi State Medical Association,
Copied contents to your clipboard!