The treatment of systemic lupus erythematosus. 1994

D Alarcón-Segovia
Department of Rheumatology, Universidad Nacional Autónoma de México, Mexico City.

Because no two SLE patients are identical, the treatment of this disease should be individualized. SLE patients, however, can be grouped by subsets, either throughout the course of the disease or at certain episodes. A number of syndromes, such as antiphospholipid or Sjögren's, also occur within SLE and may require distinct forms of treatment. The intensity of treatment in each circumstance depends on its severity and its life threatening potential. When two manifestations coexist the level of treatment usually required is determined by the most serious one. At present, the ultimate goal of treatment is achieving remission, which we define as a period of at least a year without disease activity and thus requiring no treatment. About one fourth of SLE patients achieve remission lasting a median of over 5 years, with more than half of them remaining in remission indefinitely. Current treatment of SLE is based mostly on some form of generalized immune suppression and takes little into account what is known about immune regulation in SLE. Future treatment of SLE should be tailor-made and could include use of various monoclonal antibodies, gene therapy, the administration of biological response modifiers, or the use of various hormones that may influence immune system reactivity. Also, the use of anti-idiotypic antibodies, affinity columns or injectable anionic compounds to elute or distract autoantibodies are being considered. Some of these forms of therapy are at hand but require in their application the judicious collaboration of clinicians and investigators.

UI MeSH Term Description Entries
D007166 Immunosuppressive Agents Agents that suppress immune function by one of several mechanisms of action. Classical cytotoxic immunosuppressants act by inhibiting DNA synthesis. Others may act through activation of T-CELLS or by inhibiting the activation of HELPER CELLS. While immunosuppression has been brought about in the past primarily to prevent rejection of transplanted organs, new applications involving mediation of the effects of INTERLEUKINS and other CYTOKINES are emerging. Immunosuppressant,Immunosuppressive Agent,Immunosuppressants,Agent, Immunosuppressive,Agents, Immunosuppressive
D008180 Lupus Erythematosus, Systemic A chronic, relapsing, inflammatory, and often febrile multisystemic disorder of connective tissue, characterized principally by involvement of the skin, joints, kidneys, and serosal membranes. It is of unknown etiology, but is thought to represent a failure of the regulatory mechanisms of the autoimmune system. The disease is marked by a wide range of system dysfunctions, an elevated erythrocyte sedimentation rate, and the formation of LE cells in the blood or bone marrow. Libman-Sacks Disease,Lupus Erythematosus Disseminatus,Systemic Lupus Erythematosus,Disease, Libman-Sacks,Libman Sacks Disease
D004359 Drug Therapy, Combination Therapy with two or more separate preparations given for a combined effect. Combination Chemotherapy,Polychemotherapy,Chemotherapy, Combination,Combination Drug Therapy,Drug Polytherapy,Therapy, Combination Drug,Chemotherapies, Combination,Combination Chemotherapies,Combination Drug Therapies,Drug Polytherapies,Drug Therapies, Combination,Polychemotherapies,Polytherapies, Drug,Polytherapy, Drug,Therapies, Combination Drug
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000893 Anti-Inflammatory Agents Substances that reduce or suppress INFLAMMATION. Anti-Inflammatory Agent,Antiinflammatory Agent,Agents, Anti-Inflammatory,Agents, Antiinflammatory,Anti-Inflammatories,Antiinflammatories,Antiinflammatory Agents,Agent, Anti-Inflammatory,Agent, Antiinflammatory,Agents, Anti Inflammatory,Anti Inflammatories,Anti Inflammatory Agent,Anti Inflammatory Agents

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