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In the present investigation, it was found that the number of beta-endorphin (beta-END)-immunoreactive neurons of hypothalamic arcuate nucleus (ARC) in MSG-treated rats decreased by 60.7%. No significant differences in the baseline pain threshold and the baseline corticosterone (CS) level were observed between the MSG-treated group and the control group. After electrical foot shock for 30 min, the analgesia effect was decreased significantly (P < 0.001) in MSG-treated rats, while the CS levels of both MSG-treated rats and control rats were increased remarked (P < 0.01). The results suggest that the beta-END neurons of ARC were involved in analgesia, but not in CS reaction during stress.
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
January 1985,
Peptides,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
September 1982,
Brain research,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
December 1982,
Endocrinology,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
November 1981,
The Journal of endocrinology,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
January 2000,
Physiological research,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
January 2005,
Neurotoxicology and teratology,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
May 1993,
The Journal of pharmacology and experimental therapeutics,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
February 1990,
Brain research bulletin,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
November 2005,
Neurotoxicity research,
Q L Chen, and
J H Li, and
H C Liu, and
F Gu, and
Q Z Yin
April 1997,
Pharmacology, biochemistry, and behavior,
