BIOMAT Database Logo BIOMAT Database Logo

[Diabetic coma and hyperamylasemia]. 1975

B B Moller, and K Kolendorf

UI MeSH Term Description Entries
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D010195 Pancreatitis INFLAMMATION of the PANCREAS. Pancreatitis is classified as acute unless there are computed tomographic or endoscopic retrograde cholangiopancreatographic findings of CHRONIC PANCREATITIS (International Symposium on Acute Pancreatitis, Atlanta, 1992). The two most common forms of acute pancreatitis are ALCOHOLIC PANCREATITIS and gallstone pancreatitis. Acute Edematous Pancreatitis,Acute Pancreatitis,Pancreatic Parenchyma with Edema,Pancreatic Parenchymal Edema,Pancreatitis, Acute,Pancreatitis, Acute Edematous,Peripancreatic Fat Necrosis,Acute Edematous Pancreatitides,Acute Pancreatitides,Edema, Pancreatic Parenchymal,Edematous Pancreatitides, Acute,Edematous Pancreatitis, Acute,Fat Necrosis, Peripancreatic,Necrosis, Peripancreatic Fat,Pancreatic Parenchymal Edemas,Pancreatitides, Acute,Pancreatitides, Acute Edematous,Parenchymal Edema, Pancreatic,Peripancreatic Fat Necroses
D003926 Diabetic Coma A state of unconsciousness as a complication of diabetes mellitus. It occurs in cases of extreme HYPERGLYCEMIA or extreme HYPOGLYCEMIA as a complication of INSULIN therapy. Coma, Diabetic,Comas, Diabetic,Diabetic Comas
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D006943 Hyperglycemia Abnormally high BLOOD GLUCOSE level. Postprandial Hyperglycemia,Hyperglycemia, Postprandial,Hyperglycemias,Hyperglycemias, Postprandial,Postprandial Hyperglycemias
D000328 Adult A person having attained full growth or maturity. Adults are of 19 through 44 years of age. For a person between 19 and 24 years of age, YOUNG ADULT is available. Adults
D000681 Amylases A group of amylolytic enzymes that cleave starch, glycogen, and related alpha-1,4-glucans. (Stedman, 25th ed) EC 3.2.1.-. Diastase,Amylase
D016883 Diabetic Ketoacidosis A life-threatening complication of diabetes mellitus, primarily of TYPE 1 DIABETES MELLITUS with severe INSULIN deficiency and extreme HYPERGLYCEMIA. It is characterized by KETOSIS; DEHYDRATION; and depressed consciousness leading to COMA. Acidosis, Diabetic,DKA Diabetic Ketoacidosis,Diabetic Acidosis,Diabetic Ketosis,Ketoacidosis, Diabetic,Ketosis, Diabetic,Acidoses, Diabetic,DKA Diabetic Ketoacidoses,Diabetic Acidoses,Diabetic Ketoacidoses,Diabetic Ketoacidosis, DKA,Diabetic Ketoses,Ketoacidoses, Diabetic,Ketoacidosis, DKA Diabetic,Ketoses, Diabetic

Related Publications

B B Moller, and K Kolendorf
Interpretation of hyperamylasemia in diabetic coma.
April 1973, Clinical chemistry,
B B Moller, and K Kolendorf
Reinterpretation of hyperamylasemia in diabetic coma.
June 1974, Clinical chemistry,
B B Moller, and K Kolendorf
Letter: Hyperamylasemia and diabetic ketoacidosis.
July 1974, Gastroenterology,
B B Moller, and K Kolendorf
Prolonged hyperamylasemia in diabetic ketoacidosis.
August 1976, JAMA,
B B Moller, and K Kolendorf
[Diabetic coma and hypoglycemic coma].
July 1968, Naika. Internal medicine,
B B Moller, and K Kolendorf
[DIABETIC COMA AND HYPOGLYCEMIC COMA].
April 1964, [Sogo rinsho] Clinic all-round,
B B Moller, and K Kolendorf
[Diabetic coma and insulin coma].
October 1953, La Semana medica,
B B Moller, and K Kolendorf
[DIABETIC COMA, HEPATIC COMA AND UREMIC COMA].
September 1963, Sogo igaku. Medicine,
B B Moller, and K Kolendorf
[Diabetic coma. Pathophysiology and treatment of diabetic coma].
November 1977, ZFA. Zeitschrift fur Allgemeinmedizin,
B B Moller, and K Kolendorf
Hyperamylasemia in diabetic ketoacidosis: sources and significance.
August 1979, Annals of internal medicine,
Copied contents to your clipboard!