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No significant change in hypothalamic TRH content was found in rats during acute (5-240 min) exposure to cold (5 degrees C), in spite of rapid and sustained elevations in plasma TSH and thyroxine. Plasma PRL rose markedly in the first 15 min, but returned to normal thereafter. Chronic exposure to cold (32 days) was characterized by elevated plasma and pituitary levels of both TSH and PRL in the presence of an unaltered hypothalamic TRH content. If increased TRH release from the hypothalamus occurs during exposure to cold, as suggested by the pituitary-thyroid stimulation, either it is compensated for by an equal rise in synthesis, or the extra amount released is negligible in comparison with the hypothalamic content of TRH. The acute PRL response to exposure to cold may be related to an acute TRH release but could also result from the accompanying stress response acting by a mechanism independent of TRH.
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
January 1976,
Endocrine research communications,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
January 1979,
Hormone research,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
April 1985,
Psychiatry research,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
October 1988,
Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
January 1980,
Biology of the neonate,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
August 1976,
Endocrinology,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
January 1982,
Neuroendocrinology,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
March 1974,
Acta physiologica Scandinavica,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
August 1976,
Nihon Naibunpi Gakkai zasshi,
M Jobin, and
L Ferland, and
J Côté, and
F Labrie
February 1977,
The Journal of clinical endocrinology and metabolism,
