Concentration and distribution of [3H]-SCH23390 and [3H]-YM09151-2 binding sites in midgestational human fetal cortex. 1993

A S Unis
Department of Psychiatry and Behavioral Sciences, University of Washington, Seattle 98195.

Binding sites for the dopamine receptor antagonists [3H]-SCH23390 and [3H]-YM09151-2 have been demonstrated in human fetal forebrain at the sixth gestational week and appear to increase in concentration in an age-related fashion throughout the first trimester. Using the techniques of receptor autoradiography with midsecond trimester human fetal cortex (gestational weeks 17 and 20), the distribution and concentration of binding sites for these radiolabeled ligands were compared. Saturable [3H]-SCH23390 specific binding was demonstrated in the cortical plate of the tissue obtained at gestational week 17 (KD = 0.16 nM, BMAX = 0.005 fmol/mm2). Although saturable binding for [3H]-SCH23390 could be demonstrated in the cortical plate at gestational week 20 (KD = 2.35 nM, BMAX = 0.008 fmol/mm2), image subtraction revealed specific binding in the intermediate zone as well. Saturable [3H]-YM09151-2 specific binding was likewise demonstrated in the cortical plate at gestational week 17 (KD = 0.36 nM, BMAX = 0.044 fmol/mm2). At gestational week 20, specific and saturable [3H]-YM09151-2 binding appeared to be stratified in the subplate zone, between the cortical plate and the intermediate zone (KD = 2.02 nM, BMAX = 0.076 fmol/mm2). These data suggest that specific, saturable binding for dopamine receptor antagonists can be demonstrated in human cortex by the completion of corticogenesis and at the earliest stages of cortical differentiation. The cells that express these binding sites may play a role in developing forebrain dopaminergic neuronal systems, the disturbance of which may be relevant to adult psychopathology.

UI MeSH Term Description Entries
D011247 Pregnancy The status during which female mammals carry their developing young (EMBRYOS or FETUSES) in utero before birth, beginning from FERTILIZATION to BIRTH. Gestation,Pregnancies
D011262 Pregnancy Trimester, Second The middle third of a human PREGNANCY, from the beginning of the 15th through the 28th completed week (99 to 196 days) of gestation. Midtrimester,Pregnancy, Second Trimester,Trimester, Second,Midtrimesters,Pregnancies, Second Trimester,Pregnancy Trimesters, Second,Second Pregnancy Trimester,Second Pregnancy Trimesters,Second Trimester,Second Trimester Pregnancies,Second Trimester Pregnancy,Second Trimesters,Trimesters, Second
D011954 Receptors, Dopamine Cell-surface proteins that bind dopamine with high affinity and trigger intracellular changes influencing the behavior of cells. Dopamine Receptors,Dopamine Receptor,Receptor, Dopamine
D002540 Cerebral Cortex The thin layer of GRAY MATTER on the surface of the CEREBRAL HEMISPHERES that develops from the TELENCEPHALON and folds into gyri and sulci. It reaches its highest development in humans and is responsible for intellectual faculties and higher mental functions. Allocortex,Archipallium,Cortex Cerebri,Cortical Plate,Paleocortex,Periallocortex,Allocortices,Archipalliums,Cerebral Cortices,Cortex Cerebrus,Cortex, Cerebral,Cortical Plates,Paleocortices,Periallocortices,Plate, Cortical
D005260 Female Females
D005333 Fetus The unborn young of a viviparous mammal, in the postembryonic period, after the major structures have been outlined. In humans, the unborn young from the end of the eighth week after CONCEPTION until BIRTH, as distinguished from the earlier EMBRYO, MAMMALIAN. Fetal Structures,Fetal Tissue,Fetuses,Mummified Fetus,Retained Fetus,Fetal Structure,Fetal Tissues,Fetus, Mummified,Fetus, Retained,Structure, Fetal,Structures, Fetal,Tissue, Fetal,Tissues, Fetal
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D001549 Benzamides BENZOIC ACID amides.
D001552 Benzazepines Compounds with BENZENE fused to AZEPINES.
D017447 Receptors, Dopamine D1 A subfamily of G-PROTEIN-COUPLED RECEPTORS that bind the neurotransmitter DOPAMINE and modulate its effects. D1-class receptor genes lack INTRONS, and the receptors stimulate ADENYLYL CYCLASES. Dopamine D1 Receptors,Dopamine-D1 Receptor,D1 Receptors, Dopamine,Dopamine D1 Receptor,Receptor, Dopamine-D1
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