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Patients with gastroduodenal ulcer, chronic gastritis (CG) and healthy controls (124, 57 and 24 subjects, respectively) were examined for intensity and duration of duodenogastric reflux (DGR). These indices were assessed by concentrations of gastric juice bilirubin. It is found that DGR occurred in 29% of the examinees free of gastroduodenal diseases, does not correlate with morphological CG-induced changes and Helicobacter pylori contamination degree. The reflux in CG seems to be secondary in pathogenesis of this disease. In patients with aggravated duodenal and gastric ulcer the reflux in the basal stage is 2-2.5 and 5-6 times higher than in the controls, respectively. DGR parameters correlated with the height of the ulcer defect. Retrograde entrance of the duodenal content into the stomach in duodenal ulcer enhances acid production in the stomach through gastrin mechanisms.
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
February 1994,
Clinical nuclear medicine,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
January 1988,
Lancet (London, England),
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
January 1992,
Khirurgiia,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
February 1984,
Klinicheskaia meditsina,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
January 1987,
Acta gastroenterologica Latinoamericana,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
October 1988,
Vrachebnoe delo,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
January 1981,
Scandinavian journal of gastroenterology. Supplement,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
January 1988,
Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
November 1978,
Klinicheskaia meditsina,
A S Danilov, and
O V Reshetnikov, and
S A Kurilovich
March 1990,
Khirurgiia,
