[Genetic, molecular, and immunologic aspects of Vibrio cholerae infection]. 1993

A E Fica, and F C Cabello
Department of Microbiology and Immunology, New York Medical College, Valhalla.

The deterioration of the economical and social conditions of the majority of the population in the Americas the last 20 years has generated several epidemics of enteric infections in the region, dramatically manifested by the current massive and widespread cholera outbreak. The absence of cholera from the continent for more than 100 years, the worsening environmental conditions, the biological peculiarities of Vibrio cholerae El Tor such as decreased virulence, which generates increased number of carriers, and its improved ability to thrive in the environment are probably responsible for the rapid dissemination of the disease through out the continent. Genetic and molecular studies of the biology of V cholerae have permitted identification of a variety of new virulence factors besides the enterotoxin, and are also helping to unravel the exquisite mechanisms that regulate the expression of these virulence factors in response to different stimuli. Molecular studies of V cholerae chromosomal and plasmid DNA, and of chromosomal and plasmid gene products, with techniques such as DNA hybridization and multilocus enzyme analysis are improving the characterization of V cholerae strains, resulting in progress in understanding their epidemiology in different communities. The non-invasive character of V cholerae infections, epidemiological and immunological studies suggest that the disease and current vaccines fail in providing an effective and long lasting immunity, and that the control of the disease in endemic areas by the use of vaccines may therefore be unfeasible. Similar studies indicate that the provision of safe drinking water, adequate sewage disposal, sufficient nutrition, and education remain the most effective measures for controlling the disease.

UI MeSH Term Description Entries
D002353 Carrier State The condition of harboring an infective organism without manifesting symptoms of infection. The organism must be readily transmissible to another susceptible host. Asymptomatic Carrier State,Asymptomatic Infection Carrier,Inapparent Infection Carrier,Presymptomatic Carrier State,Presymptomatic Infection Carrier,Super-spreader Carrier,Superspreader Carrier,Asymptomatic Carrier States,Asymptomatic Infection Carriers,Carrier State, Asymptomatic,Carrier State, Presymptomatic,Carrier States,Carrier, Super-spreader,Carrier, Superspreader,Carriers, Super-spreader,Carriers, Superspreader,Inapparent Infection Carriers,Infection Carrier, Asymptomatic,Infection Carrier, Inapparent,Infection Carrier, Presymptomatic,Presymptomatic Carrier States,Presymptomatic Infection Carriers,Super spreader Carrier,Super-spreader Carriers,Superspreader Carriers
D002771 Cholera An acute diarrheal disease endemic in India and Southeast Asia whose causative agent is VIBRIO CHOLERAE. This condition can lead to severe dehydration in a matter of hours unless quickly treated. Vibrio cholerae Infection,Choleras,Infection, Vibrio cholerae,Infections, Vibrio cholerae,Vibrio cholerae Infections
D002772 Cholera Toxin An ENTEROTOXIN from VIBRIO CHOLERAE. It consists of two major protomers, the heavy (H) or A subunit and the B protomer which consists of 5 light (L) or B subunits. The catalytic A subunit is proteolytically cleaved into fragments A1 and A2. The A1 fragment is a MONO(ADP-RIBOSE) TRANSFERASE. The B protomer binds cholera toxin to intestinal epithelial cells and facilitates the uptake of the A1 fragment. The A1 catalyzed transfer of ADP-RIBOSE to the alpha subunits of heterotrimeric G PROTEINS activates the production of CYCLIC AMP. Increased levels of cyclic AMP are thought to modulate release of fluid and electrolytes from intestinal crypt cells. Cholera Toxin A,Cholera Toxin B,Cholera Toxin Protomer A,Cholera Toxin Protomer B,Cholera Toxin Subunit A,Cholera Toxin Subunit B,Choleragen,Choleragenoid,Cholera Enterotoxin CT,Cholera Exotoxin,Cholera Toxin A Subunit,Cholera Toxin B Subunit,Procholeragenoid,Enterotoxin CT, Cholera,Exotoxin, Cholera,Toxin A, Cholera,Toxin B, Cholera,Toxin, Cholera
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D014734 Vibrio cholerae The etiologic agent of CHOLERA. Bacillus cholerae,Bacillus cholerae-asiaticae,Liquidivibrio cholerae,Microspira comma,Pacinia cholerae-asiaticae,Spirillum cholerae,Spirillum cholerae-asiaticae,Vibrio albensis,Vibrio cholera,Vibrio cholerae-asiaticae,Vibrio comma
D014774 Virulence The degree of pathogenicity within a group or species of microorganisms or viruses as indicated by case fatality rates and/or the ability of the organism to invade the tissues of the host. The pathogenic capacity of an organism is determined by its VIRULENCE FACTORS. Pathogenicity
D022121 Cholera Vaccines Vaccines or candidate vaccines used to prevent infection with VIBRIO CHOLERAE. The original cholera vaccine consisted of killed bacteria, but other kinds of vaccines now exist. Cholera Vaccine,Vaccine, Cholera,Vaccines, Cholera

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