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Clinical and laboratory data from a patient with Guillain-Barré syndrome indicated a probable etiological correlation of polyradiculitis to the intravenous administration of streptokinase. Oligoclonal IgG bands in the cerebrospinal fluid and serum were shown to be specific for streptokinase. Serum titers of streptokinase were elevated 64-fold for IgG, 16-fold for IgM, and 4-fold for IgA compared to controls. Clinical symptoms of Guillain-Barré syndrome are thought to result from streptokinase antibody complex mediated damage to the local blood-nerve barrier. The pathogenic relevance of autoantibodies to albumin and proteins of the central and peripheral nervous systems, occurring early after onset of symptoms, remains to be determined.
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
August 1990,
Acta neurologica Scandinavica,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
August 1987,
Southern medical journal,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
April 1984,
Annals of internal medicine,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
August 1985,
Annals of internal medicine,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
January 2022,
Neurology India,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
January 2010,
Internal medicine (Tokyo, Japan),
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
May 1992,
BMJ (Clinical research ed.),
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
December 2010,
Cardiology research and practice,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
February 1995,
The Medical journal of Australia,
R Kaiser, and
R Kaufmann, and
M Czygan, and
H Lang, and
C H Lücking
October 2004,
ANZ journal of surgery,
