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Four adolescent boys with Duchenne (progressive) muscular dystrophy (DMD) of 10-11 years duration and six normal boys of similar age were studied on a metabolism ward for 22 days. Sodium and potassium intake was as follows: Period I, Na 60 mEq, K 60 mEq; Period II, Na 10, K 60; Period III, Na 10, K 95-150; Period IV, Na 60, K 60. The differences between the DMD group and the group of normal boys for sodium and potassium in serum and urine and for urinary aldosterone were not significant. These findings show that the pathologically elevated sodium-potassium ratio in skeletal muscle of patients with DMD is not due to increased aldosterone or other causes of renal wastage of potassium.
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
July 1996,
Muscle & nerve,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
January 1979,
Transactions of the American Neurological Association,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
December 2011,
Neurology,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
June 1994,
Free radical biology & medicine,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
March 1976,
Archives of neurology,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
January 2020,
PloS one,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
June 1991,
Current opinion in genetics & development,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
June 1999,
Thorax,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
January 2008,
Neurology India,
J B Garst, and
P J Vignos, and
M Hadaday, and
D N Matthews
January 1995,
The Southeast Asian journal of tropical medicine and public health,
